Glutathione-gold nanoclusters enhance sweet potato thermotolerance through improved photosynthesis, redox homeostasis, and cellular integrity

✅ 全文

谷胱甘肽-金纳米簇通过改善光合作用、氧化还原稳态和细胞完整性增强甘薯耐热性

作者 Sunjeet Kumar; Gurmendar; Rui Yu; Muhammad Ikram; Jingjing Kou; Muhammad Abbas Khan; Mengzhao Wang; Guopeng Zhu 期刊 BMC Plant Biology 发表日期 2026 ISSN 1471-2229 DOI 10.1186/s12870-026-08417-w 类型 原创研究 (Original Research)

📄 英文摘要 English Abstract

EN

Global warming severely challenges sweet potato cultivation by disrupting physiological, biochemical, and structural processes. The potential of glutathione-gold nanoclusters (GSH-Au NCs) in enhancing thermotolerance remains largely unexplored in vegetable sweet potato, a key industrial and food security crop. We hypothesized that foliar application of GSH-Au NCs would mitigate heat-induced disruptions in photosynthesis, redox homeostasis, and cellular structures by enhancing antioxidant defenses and enzymatic activities. Under controlled heat stress (42 °C day/35°C night for 7 days), heat stress diminished shoot biomass, leaf area, RWC, and root biomass, while impairing photosynthesis and elevating oxidative damage (H2O2: 276.7% and MDA: 481.5%) and electrolyte leakage by 85.7%. GSH-Au NCs (2 mg L− 1) reversed these effects, enhancing growth, shoot and root biomass (45.3% and 28.3%), and RWC (7.9%). These improvements were associated with enhanced photosynthetic efficiency through elevated chlorophyll content (72.6%), Rubisco activity (26.4%), gas exchange parameters (Pn: 86.1% and Gs: 389%), and chlorophyll fluorescence (Fv/Fm: 16.0% and ETR: 48.2%). Mechanistically, GSH-Au NCs correlated with upregulation of ascorbate-glutathione (AsA-GSH) cycle, boosting enzymatic (SOD: 30.1%, APX: 33.6%, and GR: 20.9%) and non-enzymatic antioxidants (AsA/DHA: 33.8% and GSH/GSSG: 29.1%), while lowering oxidative markers: H2O2 (37.8%), MDA (51.7%), and EL (26.8%), compared with heat stressed plants without NCs treatment. Transcriptional upregulation of SOD, APX, GR, DHAR, and MDHAR genes supported these effects. Additionally, GSH-Au NCs enhanced glyoxalase activity (Gly I: 36.9% and Gly II: 35.0%), reducing toxic methylglyoxal (29.3%). Higher proline (51.7%) and secondary metabolites (polyphenols: 38.3% and flavonoids: 62.9%) further strengthen stress resilience. Moreover, GSH-Au NCs restored stomatal behavior and preserved chloroplast and mitochondrial structure. These findings highlight GSH-Au NCs (2 mg L− 1) as a sustainable, cost-effective, eco-friendly nanobiotechnological strategy for mitigating heat stress in sweet potato and promoting climate-resilient horticulture, advancing beyond prior studies on individual GSH or Au nanomaterials by demonstrating the synergetic effects of their combined nanocluster form for the first time in plant species.

📄 中文摘要 Chinese Abstract

中文
全球变暖通过破坏生理、生化和结构过程,严重挑战甘薯栽培。热胁迫的特征是温度超过植物最适生长范围,破坏细胞稳态,损害关键生理过程,降低作物产量,并对植被产生长期影响。热胁迫深刻影响植物生理,特别是损害光合效率。温度升高诱导气孔关闭,从而限制CO₂吸收并降低Rubisco羧化效率,同时促进光呼吸。关键光合酶如Rubisco失活、叶绿素降解和叶绿体结构损伤加剧了这些效应。此外,光合传递链中过量的电子流增加了活性氧(ROS)的产生,包括超氧化物、过氧化氢和甲基乙二醛。升高的ROS水平导致氧化应激、脂质过氧化和膜损伤。植物通过防御系统应对热胁迫条件,包括酶清除剂和谷胱甘肽和脯氨酸等非酶化合物。谷胱甘肽-金纳米簇(GSH-Au NCs)在增强耐热性方面的潜力在蔬菜甘薯中很大程度上尚未被探索。

📋 英文结构化总结 English Structured Summary

全文整理

EN

Header:

Background Global warming severely challenges sweet potato cultivation by disrupting physiological, biochemical, and structural processes. Heat stress is characterized by temperatures exceeding a plant’s optimal growth range, disrupting cellular homeostasis, impairing key physiological processes, and reducing crop yields, with long-term consequences for vegetation. Heat stress profoundly impacts plant physiology, particularly compromising photosynthetic efficiency. Elevated temperatures induce stomatal closure, thereby restricting CO₂ uptake and diminishing Rubisco carboxylation efficiency, which simultaneously promotes photorespiration. The inactivation of key photosynthetic enzymes like Rubisco, chlorophyll degradation, and structural damage to chloroplasts intensifies these effects. Additionally, excessive electron flow in the photosynthetic transport chain increases generations of reactive oxygen species (ROS), including superoxide, hydrogen peroxide, and methylglyoxal. Elevated ROS levels cause oxidative stress, lipid peroxidation, and membrane damage. Plants counteract heat stress conditions through defense systems, comprising enzymatic scavengers and non-enzymatic compounds like glutathione and proline. The potential of glutathione-gold nanoclusters (GSH-Au NCs) in enhancing thermotolerance remains largely unexplored in vegetable sweet potato.

Header:

Methods We hypothesized that foliar application of GSH-Au NCs would mitigate heat-induced disruptions in photosynthesis, redox homeostasis, and cellular structures by enhancing antioxidant defenses and enzymatic activities. Under controlled heat stress (42 °C day/35°C night for 7 days), heat stress diminished shoot biomass, leaf area, RWC, and root biomass, while impairing photosynthesis and elevating oxidative damage and electrolyte leakage. GSH-Au NCs (2 mg L⁻¹) reversed these effects.

Header:

Results GSH-Au NCs (2 mg L⁻¹) enhanced growth, shoot and root biomass (45.3% and 28.3%), and RWC (7.9%). These improvements were associated with enhanced photosynthetic efficiency through elevated chlorophyll content (72.6%), Rubisco activity (26.4%), gas exchange parameters (Pn: 86.1% and Gs: 389%), and chlorophyll fluorescence (Fv/Fm: 16.0% and ETR: 48.2%). Mechanistically, GSH-Au NCs correlated with upregulation of ascorbate-glutathione cycle, boosting enzymatic and non-enzymatic antioxidants, while lowering oxidative markers: H₂O₂ (37.8%), MDA (51.7%), and EL (26.8%). Transcriptional upregulation of SOD, APX, GR, DHAR, and MDHAR genes supported these effects. Additionally, GSH-Au NCs enhanced glyoxalase activity, reducing toxic methylglyoxal (29.3%). Higher proline (51.7%) and secondary metabolites (polyphenols: 38.3% and flavonoids: 62.9%) further strengthened stress resilience. Moreover, GSH-Au NCs restored stomatal behavior and preserved chloroplast and mitochondrial structure.

Header:

Data Summary Heat stress elevated H₂O₂ by 276.7% and MDA by 481.5%, and electrolyte leakage by 85.7%. GSH-Au NCs treatment reduced H₂O₂ by 37.8%, MDA by 51.7%, and EL by 26.8% compared with heat-stressed plants without NCs treatment. Enzymatic antioxidants increased: SOD 30.1%, APX 33.6%, GR 20.9%; non-enzymatic ratios improved: AsA/DHA 33.8%, GSH/GSSG 29.1%. Glyoxalase activities increased: Gly I 36.9%, Gly II 35.0%. Proline increased 51.7%, polyphenols 38.3%, flavonoids 62.9%.

Header:

Conclusions These findings highlight GSH-Au NCs (2 mg L⁻¹) as a sustainable, cost-effective, eco-friendly nanobiotechnological strategy for mitigating heat stress in sweet potato and promoting climate-resilient horticulture, advancing beyond prior studies on individual GSH or Au nanomaterials by demonstrating the synergetic effects of their combined nanocluster form for the first time in plant species.

Header:

Practical Significance GSH-Au NCs (2 mg L⁻¹) represent a sustainable, cost-effective, eco-friendly nanobiotechnological strategy for mitigating heat stress in sweet potato and promoting climate-resilient horticulture.

📋 中文结构化总结 Chinese Structured Summary

中文

背景:

全球变暖通过破坏生理、生化和结构过程,严重挑战甘薯栽培。热胁迫的特征是温度超过植物最适生长范围,破坏细胞稳态,损害关键生理过程,降低作物产量,并对植被产生长期影响。热胁迫深刻影响植物生理,特别是损害光合效率。温度升高诱导气孔关闭,从而限制CO₂吸收并降低Rubisco羧化效率,同时促进光呼吸。关键光合酶如Rubisco失活、叶绿素降解和叶绿体结构损伤加剧了这些效应。此外,光合传递链中过量的电子流增加了活性氧(ROS)的产生,包括超氧化物、过氧化氢和甲基乙二醛。升高的ROS水平导致氧化应激、脂质过氧化和膜损伤。植物通过防御系统应对热胁迫条件,包括酶清除剂和谷胱甘肽和脯氨酸等非酶化合物。谷胱甘肽-金纳米簇(GSH-Au NCs)在增强耐热性方面的潜力在蔬菜甘薯中很大程度上尚未被探索。

方法:

我们假设叶面喷施GSH-Au NCs将通过增强抗氧化防御和酶活性来缓解热胁迫对光合作用、氧化还原稳态和细胞结构的破坏。在受控热胁迫条件下(42°C白天/35°C夜间,持续7天),热胁迫降低了地上部生物量、叶面积、相对含水量和根系生物量,同时损害光合作用并增加氧化损伤和电解质渗漏。GSH-Au NCs(2 mg L⁻¹)逆转了这些效应。

结果:

GSH-Au NCs(2 mg L⁻¹)增强了生长,地上部和根系生物量分别增加45.3%和28.3%,相对含水量增加7.9%。这些改善与光合效率的提高相关,表现为叶绿素含量增加72.6%,Rubisco活性增加26.4%,气体交换参数(净光合速率增加86.1%和气孔导度增加389%),以及叶绿光化学效率(Fv/Fm增加16.0%和电子传递速率增加48.2%)。机制上,GSH-Au NCs与抗坏血酸-谷胱甘肽循环的上调相关,增强了酶促和非酶促抗氧化剂,同时降低了氧化标志物:H₂O₂降低37.8%,丙二醛降低51.7%,电解质渗漏降低26.8%。SOD、APX、GR、DHAR和MDHAR基因的转录上调支持了这些效应。此外,GSH-Au NCs增强了乙二醛酶活性,降低了有毒甲基乙二醛(29.3%)。较高的脯氨酸(51.7%)和次生代谢物(多酚增加38.3%和类黄酮增加62.9%)进一步增强了抗逆性。此外,GSH-Au NCs恢复了气孔行为并保护了叶绿体和线粒体结构。

数据总结:

热胁迫使H₂O₂升高276.7%,丙二醛升高481.5%,电解质渗漏升高85.7%。与未处理的胁迫植株相比,GSH-Au NCs处理使H₂O₂降低37.8%,丙二醛降低51.7%,电解质渗漏降低26.8%。酶促抗氧化剂增加:SOD增加30.1%,APX增加33.6%,GR增加20.9%;非酶促比率改善:抗坏血酸/脱氢抗坏血酸比率增加33.8%,还原型谷胱甘肽/氧化型谷胱甘肽比率增加29.1%。乙二醛酶活性增加:Gly I增加36.9%,Gly II增加35.0%。脯氨酸增加51.7%,多酚增加38.3%,类黄酮增加62.9%。

结论:

这些发现突出了GSH-Au NCs(2 mg L⁻¹)作为一种可持续、经济、环保的纳米生物技术策略,用于缓解甘薯热胁迫并促进气候适应性园艺,通过首次证明其组合纳米簇形式的协同效应,超越了先前对单独GSH或金纳米材料的研究。

实际意义:

GSH-Au NCs(2 mg L⁻¹)代表了一种可持续、经济、环保的纳米生物技术策略,用于缓解甘薯热胁迫并促进气候适应性园艺。

📖 英文全文 English Full Text

EN

59 bmcps BMC Plant Biology BMC Plant Biol BMC PMC13059601 13059601 13059601 41772431 10.1186/s12870-026-08417-w Glutathione-gold nanoclusters enhance sweet potato thermotolerance through improved photosynthesis, redox homeostasis, and cellular integrity Kumar Sunjeet 1 2 # Gurmendar 1 2 # Yu Rui 1 2 Ikram Muhammad 1 2 Kou Jingjing 1 2 Khan Muhammad Abbas 1 2 Wang Mengzhao 1 2 ✉ Zhu Guopeng 1 2 ✉ 1 School of Breeding and Multiplication (Sanya Institute of Breeding and Multiplication), Hainan University, Sanya, 572025 China 2 Key Laboratory for Quality Regulation of Tropical Horticultural Crops of Hainan Province, School of Tropical Agriculture and Forestry, Hainan University, Haikou, 570228 China ✉ Corresponding author. # Contributed equally. 2 3 2026 26 627 627 9 4 2026 © The Author(s) 2026 Open Access This article is licensed under a Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 International License, which permits any non-commercial use, sharing, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if you modified the licensed material. You do not have permission under this licence to share adapted material derived from this article or parts of it. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by-nc-nd/4.0/ . Abstract Background Global warming severely challenges sweet potato cultivation by disrupting physiological, biochemical, and structural processes. The potential of glutathione-gold nanoclusters (GSH-Au NCs) in enhancing thermotolerance remains largely unexplored in vegetable sweet potato, a key industrial and food security crop. We hypothesized that foliar application of GSH-Au NCs would mitigate heat-induced disruptions in photosynthesis, redox homeostasis, and cellular structures by enhancing antioxidant defenses and enzymatic activities. Results Under controlled heat stress (42 °C day/35°C night for 7 days), heat stress diminished shoot biomass, leaf area, RWC, and root biomass, while impairing photosynthesis and elevating oxidative damage (H 2 O 2 : 276.7% and MDA: 481.5%) and electrolyte leakage by 85.7%. GSH-Au NCs (2 mg L − 1 ) reversed these effects, enhancing growth, shoot and root biomass (45.3% and 28.3%), and RWC (7.9%). These improvements were associated with enhanced photosynthetic efficiency through elevated chlorophyll content (72.6%), Rubisco activity (26.4%), gas exchange parameters (Pn: 86.1% and Gs: 389%), and chlorophyll fluorescence (Fv/Fm: 16.0% and ETR: 48.2%). Mechanistically, GSH-Au NCs correlated with upregulation of ascorbate-glutathione (AsA-GSH) cycle, boosting enzymatic (SOD: 30.1%, APX: 33.6%, and GR: 20.9%) and non-enzymatic antioxidants (AsA/DHA: 33.8% and GSH/GSSG: 29.1%), while lowering oxidative markers: H 2 O 2 (37.8%), MDA (51.7%), and EL (26.8%), compared with heat stressed plants without NCs treatment. Transcriptional upregulation of SOD , APX , GR , DHAR , and MDHAR genes supported these effects. Additionally, GSH-Au NCs enhanced glyoxalase activity (Gly I: 36.9% and Gly II: 35.0%), reducing toxic methylglyoxal (29.3%). Higher proline (51.7%) and secondary metabolites (polyphenols: 38.3% and flavonoids: 62.9%) further strengthen stress resilience. Moreover, GSH-Au NCs restored stomatal behavior and preserved chloroplast and mitochondrial structure. Conclusions These findings highlight GSH-Au NCs (2 mg L − 1 ) as a sustainable, cost-effective, eco-friendly nanobiotechnological strategy for mitigating heat stress in sweet potato and promoting climate-resilient horticulture, advancing beyond prior studies on individual GSH or Au nanomaterials by demonstrating the synergetic effects of their combined nanocluster form for the first time in plant species. Supplementary Information The online version contains supplementary material available at 10.1186/s12870-026-08417-w. Keywords: Thermotolerance, GSH-Au NCs, Redox balance, Photosynthetic efficiency, AsA-GSH cycle, Ultrastructure status released display-pdf yes is-olf no is-manuscript no is-preprint no is-journal-matter no is-scanned no is-retracted no Received 2025 Dec 29; Accepted 2026 Feb 17; Collection date 2026. Introduction Climate change-driven global warming poses a severe threat to plant health and agricultural productivity, as increasing temperatures and frequent heatwaves negatively affect crop production [ 1 , 2 ]. Heat stress is characterized by temperatures exceeding a plant’s optimal growth range, disrupting cellular homeostasis, impairing key physiological processes, and reducing crop yields, with long-term consequences for vegetation [ 2 , 3 ]. Studies predict a continued rise in global temperatures, potentially decreasing crop yields by 3–8% for every 1 °C increase, particularly in tropical and subtropical regions where 70% of the global population resides [ 4 , 5 ]. As a leading agricultural producer, China faces a growing threat from rising heatwaves, which endanger food security by damaging critical processes in crops, such as photosynthesis, water regulation, and cellular integrity [ 6 – 8 ]. Heat stress profoundly impacts plant physiology, particularly compromising photosynthetic efficiency [ 9 ]. Elevated temperatures induce stomatal closure, thereby restricting CO 2 uptake and diminishing Rubisco carboxylation efficiency, which simultaneously promotes photorespiration [ 10 ]. While stomatal closure helps conserve water, it also exacerbates CO 2 limitation, further reducing photosynthetic efficiency. The inactivation of key photosynthetic enzymes like Rubisco, chlorophyll degradation, and structural damage to chloroplasts intensifies these effects. Additionally, excessive electron flow in the photosynthetic transport chain, particularly between photosystem II (PSII) and PSI, increases generations of reactive oxygen species (ROS), including superoxide (O 2 •− ), hydrogen peroxide (H 2 O 2 ), and methylglyoxal (MG). Elevated ROS levels cause oxidative stress, lipid peroxidation, and membrane damage [ 6 , 10 , 11 ]. Malondialdehyde (MDA) is a marker of lipid peroxidation, increases under heat stress, indicating damage to cellular structures such as chloroplasts and mitochondria. Furthermore, restricted root respiration and phloem transport hinder sugar translocation, causing carbohydrate accumulation in leaves and further suppressing CO 2 assimilation [ 8 , 10 ]. Plants counteract heat stress conditions through defense systems, comprising enzymatic scavengers, including superoxide dismutase (SOD), catalase (CAT), peroxidase (POD), and ascorbate peroxidase (APX), along with non-enzymatic compounds like glutathione (GSH) and proline. The ascorbate-glutathione (AsA-GSH) cycle is critical for ROS detoxification, neutralizing H 2 O 2 using AsA as an electron donor, producing monodehydroascorbate (MDHAR) and dehydroascorbate (DHAR). GR regenerates GSH from GSSG using NADPH, sustaining the cycle’s efficiency [ 12 , 13 ]. Additionally, the toxic compound MG is detoxified through the coordinated action of glyoxalase (Gly I and II) [ 11 ]. These defense mechanisms enable heat-tolerant cultivars to maintain cellular redox balance and protect their photosynthetic machinery, while sensitive cultivars often experience severe cellular damage and accelerated premature leaf senescence [ 3 ]. Prolonged stress exhausts the antioxidant defense system, rendering it ineffective. Applying exogenous growth regulators can then improve stress tolerance and recovery. Conventional methods to mitigate heat stress, such as chemical treatments and agronomic practices, often present limitations such as environmental toxicity, high costs, and risks to soil microbial communities [ 14 ]. In contrast, nanotechnology has emerged as a promising eco-friendly solution, which provides improved nutrient delivery, enhanced stress tolerance, and sustainable crop protection [ 15 , 16 ]. Various nanomaterials (NMs) like nanoclusters (NCs) and nanoparticles (NPs), including Au, SiO 2 , CeO 2 , and MT-Se, improve plant stress tolerance by enhancing biomass accumulation, nutrient uptake, photosynthetic efficiency, and ROS scavenging [ 17 – 20 ]. These nanomaterials influence cellular structure, physiological processes, and gene expression related to aquaporins, photosynthesis, and antioxidant defense [ 21 , 22 ]. Glutathione (GSH), a key regulator of detoxification, cell division, and stress tolerance, enhances antioxidant enzyme activity and protects photosynthetic processes under heat and other abiotic stress conditions [ 23 , 24 ]. While, gold (Au) nanomaterials and GSH supplementation have individually shown promise in enhancing plant stress tolerance, the combined application of glutathione-gold nanoclusters (GSH-Au NCs) has not yet been reported in any plant species. GSH-Au NCs were selected for their potential synergetic effects, where GSH’s antioxidant and detoxification roles such as ROS scavenging and AsA-GSH cycle regulation are enhanced by Au nanoclusters’ biophysical properties, such as high surface area for efficient delivery, free radical scavenging, and upregulation of stress response genes, as observed in previous Au NP studies [ 25 – 27 ]. This combination aims to provide a more targeted and stable internation compared to individual components. This study addresses key gaps in prior nano-enabled heat-stress mitigation research, which has focused on individual nanomaterials such as Au NPs and GSH alone in different crops [ 17 – 20 ], but lacks combined GSH-Au NC formulations, applications in vegetable sweet potato, and detailed mechanistic insights into ultrastructural, transcriptional, and glyoxalase responses. Sweet potato ( Ipomoea batatas ) was selected due to its high sensitivity to heat stress such as reduced biomass and yield at temperature more than 35 °C [ 13 , 28 , 29 ]. Sweet potato has agronomical importance as the world’s 7th most economically important starch crop (global production of 93.5 million metric tons annually, and China producing 55% of global output alone) FAO, 2023, https://www.fao.org/faostat/en/#data/QCL/visualize ). Previous studies on sweet potato under heat stress have primarily focused on gene expression, storage root development, and genetic transformation, with limited research on vegetable sweet potatoes [ 28 – 31 ], and also the absence of previous nanomaterial-based stress mitigation studies in this species under heat stress. Therefore, we hypothesize that foliar application of GSH-Au NCs will enhance thermotolerance in sweet potato by (i) improving photosynthetic efficiency through increased pigment content, Rubisco activity, and gas exchange, (ii) strengthening redox balance via activation of the AsA-GSH cycle, glyoxalase system, antioxidant defenses, and related gene expression, and (iii) preserving structural integrity, including stomatal behavior and chloroplast ultrastructure. The findings aim to establish GSH-Au NCs as a cost-effective, eco-friendly, and sustainable approach to improve sweet potato growth and tolerance under heat stress, offering novel insights for climate-resilient agriculture. Materials and methods Characterization of purchased nanoclusters Analytical-grade GSH-Au NCs were obtained from XFNANO Materials Technology Co., Ltd. (Jiangsu, China) with a purity of higher than 99.5%. GSH-Au NCs were characterized using HR-TEM (S-4800, Hitachi, Tokyo, Japan). The nanomaterial suspension was ultrasonicated for 30 min in the dark, after which zeta potential, UV-visible absorption spectra, and particle size distribution were calculated using a Xetasizer Nano ZS90 (Malvern Instruments, Marburg, UK) [ 6 ]. GSH-Au NCs exhibited spherical morphology with an average < 3 nm (HR-TEM), positive zeta potential (22.7 ± 1.2 mV, confirming colloidal stability), and a UV-visible absorption peak at 418 nm (Fig. 1 ; S1). Fig. 1 Characterization of glutathione-gold nanoclusters (GSH-Au NCs) and growth parameters under normal and heat stress conditions. A HR-TEM image of GSH-Au NCs (scale bar: 2 nm), B zeta potential of GSH-Au NCs, C representative phenotypes of vegetable sweet potato under different treatments (CK: control, GA: 2 mg L − 1 GSH-Au NCs, HS: heat stress, GAHS: 2 mg L − 1 GSH-Au NCs + heat stress). and D-K) quantitative growth parameters: D plant height (cm plant − 1 ), E leaf area (cm 2 ), F number of leaves plant − 1 , G relative water content, H shoot fresh weight (g plant − 1 ), I shoot dry weight (g plant − 1 ), J root fresh weight (g plant − 1 ), and ( K ) root dry weight (g plant − 1 ). Data are mean ± SE from biological triplicates. Different letters indicate significant differences (Duncan’s test, P  < 0.05) Preparation of plant materials and experimental design The experiments used the sweet potato cultivar ‘HD7791’, which was selected and evaluated by our research group from the cultivated population in Hainan Province, China. Planting material (stem cuttings) for this study was sourced from our institutional stock. The cultivar’s identity was formally confirmed by our research team, and a resource is maintained at Hainan University. Stem cuttings were disinfected with fungicide and grown in soil-filled pots at 25–27 °C with a 16-h photoperiod. After a 7-day acclimatization period, the growth chamber was set to 42 °C (light) and 35 °C (dark), with 4,000 lx light intensity (80 µmol m −2 s −1 PPFD, based on LED conversion factors), a 16/8-h photoperiod, and 75% humidity. This moderate light level was selected to maintain consistent vegetative conditions during the 7-day acute heat stress exposure, allowing clear detection of heat-induced physiological disruptions (photosynthesis decline and oxidative damage) without confounding by high-light photoinhibition. This regime mimics extreme heatwaves in tropical and subtropical sweet potato growing regions, where summer temperatures often exceed 42 °C, causing 20–40% yield losses [ 4 , 5 , 28 ]. Foliar applications were initiated at a defined early vegetative stage (7 days post-acclimatization), when initial heat stress phenotypes such as mild wilting emerged uniformly across plants in pilot trials, ensuing consistent timing and minimizing bias. The selected concentration of GSH-Au NCs (0, 0.5, 1, 2, and 4 mg L −1 ) was based on preliminary optimization and concentrations previously reported as physiologically effective for nanomaterial-mediated stress mitigation without phytotoxic effects. The experimental design included the following treatments: (1) CK; Control, (2) GA1; 0.5 mg L −1 GSH-Au NCs, (3) GA2; 1 mg L −1 GSH-Au NCs, (4) GA3; 2 mg L −1 GSH-Au NCs, (5) GA4; 4 mg L −1 GSH-Au NCs, (6) HS; heat stress 42 °C, (7) GAHS1; 0.5 mg L −1 GSH-Au NCs + heat stress, (8) GAHS2; 1 mg L −1 GSH-Au NCs + heat stress, (9) GAHS3; 2 mg L −1 GSH-Au NCs + heat stress, and (10) GAHS4; 4 mg L −1 GSH-Au NCs + heat stress. Phenotypic and physiological data were recorded after 7 days of NC treatment. Determination of plant growth parameters Growth parameters of sweet potato seedlings, including height, leaf number, leaf area, and fresh and dry weights of roots and shoots, were evaluated. Leaf area was determined with a portable laser leaf area meter (CI-202, CID Bio-Science, USA). Fresh weights (FW) were reordered, followed by drying at 80 °C for 72 h to determine dry weights (DW). Relative water content (RWC) was calculated using established methods [ 24 ]. After determining the FW of the leaves, we immersed them in ddH 2 O for 4 h and then determined their turgor weight (TW). The leaves were then desiccated at 70 °C for 24 h. Finally, the RWC of the leaves was measured using the following formula; RWC (%) = [(FW-DW)/(TW-DW)] × 100 After washing, fresh roots were scanned using an Epson Expression 11000XL root scanner to determine root morphology. Images were analyzed using WinRHIZO 2003a software, a validated and reliable tool for root traits in recent plant studies [ 32 ]. Determination of photosynthesis-related parameters Leaf gas exchange parameters were measured using a portable photosynthesis system. Chlorophyll and carotenoid contents were determined by homogenizing 0.1 g of fresh leaves in 80% acetone, followed by centrifugation and absorbance measurement at 662, 645, and 470 nm. Chlorophyll and carotenoid visualization was performed using a TCS SP2 laser confocal microscope (Leica, Germany). A portable chlorophyll fluorometer was used to quantify ETR, qP, qN, Y(II), and Fv/Fm [ 32 , 33 ]. Electron microscopic analysis For scanning electron microscopy (SEM), fresh leaves were rinsed with distilled water, fixed in glutaraldehyde, and dehydrated in 80% ethanol. Samples were then dried using critical point drying, fixed on SEM stubs with conductive tape, and coated with platinum through sputtering for 10 min before SEM imaging [ 34 ]. For transmission electron microscopy (TEM), fresh leaf tissues were dissected into small pieces and fixed in 2.5% glutaraldehyde at 4 °C for 12 h. Samples were rinsed three times in 0.1 M phosphate-buffered saline (PBS, pH 7.4) for 15 min each, post-fixed in 1% osmium tetroxide for 1–2 h, and washed again three times in PBS for 15 min each. Dehydration was performed using a graded ethanol series (30%, 50%, 70%, and 90% for 15 min each, followed by 100% ethanol twice for 20 min each) and two changes of 100% acetone for 20 min each. Infiltration was carried out with acetone: embedding resin mixtures (1:1 at 37 °C for 3 h and 1:3 at 37 °C for 4 h), followed by pure embedding resin overnight at 37 °C. Samples were subsequently embedded in fresh resin molds and polymerized at 70 °C for 12–48 h. Ultrathin sections were cut, mounted on copper grids, stained with uranyl acetate for 8–15 min followed by lead citrate for 5–10 min, air-dried, and examined under a HITACHI HT7800 TEM [ 24 ]. Oxidative damage detection by histochemical localization H 2 O 2  and O 2 •− were localized using DAB and NBT staining, respectively. MDA was detected by staining fresh leaves with 10% Schiff’s reagent for 120 min, followed by washing in potassium metabisulfite solution to remove residual stain [ 35 ]. Determination of oxidative stress markers, antioxidants, and antioxidant enzymes Electrolyte leakage (EL) was measured using a previously established protocol [ 36 ]. Levels of oxidative stress markers (H 2 O 2 , O 2 •− , and MDA), antioxidants (AsA, DHA, GSH, and GSSG), Rubisco activity, and antioxidant enzyme activities (SOD, CAT, APX, GR, DHAR, MDHAR, POD, and GST) were quantified using commercially available kits from Nanjing Jiancheng Bioengineering and Solarbio. Leaf samples were homogenized, centrifuged, and the supernatants were analyzed following kit instructions [ 13 , 24 ]. MG content and activities of Gly I and Gly II were measured using a previously described protocol [ 36 ]. Determination of osmolytes and secondary metabolite content Soluble sugars were extracted by homogenizing leaf samples in double-distilled water, heating at 95 °C for 15 min, and centrifuging at 8,000 g for 12 min. Absorbance was measured at 620 nm using a commercial test kit from Nanjing Jiancheng Bioengineering, China. Total proteins and proline concentrations were quantified using commercial kits (A0452 and A107-1-1) following homogenization and centrifugation [ 13 , 24 ]. The Folin-Ciocalteu method was used for the determination of total polyphenols, and the aluminum chloride method was used for flavonoid content [ 24 , 37 ]. qRT-PCR analysis Total RNA was extracted from frozen leaf samples using a Trizol kit (Invitrogen, Santa Cruz, CA, USA). Reverse transcription was performed using a SuperScript III reverse transcriptase kit. The Actin gene (accession: EU250003 ) was used as the internal reference for normalization, selected based on its stable expression across treatments. Gene-specific primers were designed using Primer3 software (primer sequences listed in Table S2). qRT-PCR analysis was conducted using a Bio-Rad Mx3000P qPCR system with three biological replicates ( n = 3 independent plants per treatment). Expression levels were calculated using the 2 –∆∆ Ct method [ 38 , 39 ]. Statistical analysis All experimental measurements were conducted in biological triplicate ( n  = 3 independent plants per treatment. Data were expressed as mean ± standard error (SE). Statistical differences among treatments were analyzed using one-way ANOVA followed by Duncan’s multiple range test at P  < 0.05 in SPSS 25.0 (IBM, Chicago, USA). Duncan’s test was selected for its sensitivity in detecting differences in multi-treatment agricultural experiments, as is commonly used in plant stress studies. To minimize the risk of type I error inflation associated with Duncan’s test, all key results were cross-verified with HSD test ( P  < 0.05), which showed no discrepancies in significance patterns. Statistical power was confirmed post-hoc for major parameters (power > 0.8). Pearson’s correlation analysis was performed using R software (Package ggcor). Results Plant growth variables Different concentrations were tested under both normal and heat stress conditions to investigate the biological impact of GSH-Au NCs on sweet potato plant. Under non-stress conditions, GSH-Au NCs (GA1 to GA4) lead to significant enhancements in growth and biomass accumulation compared to control plants ( P  < 0.05), with the GA3 (2 mg L − 1 ) treatment significantly promoted growth, increasing shoot and root fresh biomass by 13.5% and 12.1% and dry biomass by 7.2% and 9.2% and photosynthetic pigments by 3.6–7.2% (Table S1; Fig.  2 ), indicating general growth-promoting effects in addition to stress mitigation. Fig. 2 Auto-fluorescence, chlorophyll and carotenoid content in the leaves of sweet potato under normal and heat stress conditions. A Auto-fluorescence of chlorophyll and carotenoids, and ( B - E ) chlorophyll and carotenoid content (mg g − 1 FW) under normal and heat stress conditions. Treatments: CK= control, GA = 2 mg L − 1 GSH-Au NCs, HS= heat stress, and GAHS = 2 mg L − 1 GSH-Au NCs + heat stress. Data are means ± SE from n  = 3 biological replicates. Different letters indicate significant differences (Duncan’s test, P  < 0.05) However, plants subjected to heat stress severely impaired plant development, reducing plant height (24.1%), shoot FW and DW (41.6 and 43.1%), leaf number (56.5%), leaf area (66.7%), RWC (15.1%), root biomass (40.4% FW and 31.5% DW) compared to the control plants (Fig.  1 , Table S1). Remarkably, GSH-Au NCs mitigated these adverse effects, with the 2 mg L − 1 (GAHS3) treatment demonstrating the highest efficacy by enhancing plant height (18.9%), shoot biomass (45.3% FW, 35.2% DW), leaf traits (100% increase in leaf number and 95.7% in leaf area), RWC (7.9%), and root biomass (28.3% FW and 25.6% DW) relative to heat-stressed plants (Fig.  1 , Table S1). Based on these results, the 2 mg L − 1 GSH-Au NCs concentration was identified as the optimal treatment and was selected for further analysis. Root system architecture was also severely affected by heat stress ( P  < 0.05; Fig. S1). However, sweet potato plants treated with 2 mg L − 1 GSH-Au NCs demonstrated notable recovery in root traits, including significant increases in root length (27.9%), surface area (68.1%), diameter (12.7%), root volume (59.9%), projected area, tips, forks, and crossings compared to heat-stressed plants (Fig. S2). Photosynthetic pigments, rubisco activity, and photosynthetic efficiency Under normal conditions, GSH-Au NCs slightly enhanced photosynthetic pigment levels and efficiency (Figs.  2 and 3 ). However, exposure to heat stress drastically decreased the level of T. Chl, Chl a, Chl b, and Car by 63.7%, 53.5%, 74.5%, and 46.6%, respectively (Fig.  2 ). While GSH-Au NCs (2 mg L − 1 ) significantly increased pigment concentrations under heat stress, with increases of 72.6% (T. Chl), 69.2% (Chl a), 79.1% (Chl b), and 28.2% (Car) compared to heat-stressed plants (Fig.  2 B-E). Confocal microscopy revealed uniform and dense red (chlorophyll) and green (carotenoids) fluorescence in both control and NC-treated plants under normal conditions. However, heat-stressed leaves exhibited a sharp decline in chlorophyll and carotenoid fluorescence. The application of GSH-Au NCs restored fluorescence intensity, aligning with the observed pigment recovery (Fig.  2 A). Fig. 3 Chlorophyll fluorescence, gas exchange parameters, and Rubisco activity of vegetable sweet potato under normal and heat stress conditions. A - E Fv/Fm, Y(II), qP, and ETR. F - I Pn, Gs, Ci, and Tr. J Rubisco activity under normal and heat stress conditions. Treatments: CK= control, GA = 2 mg L − 1 GSH-Au NCs, HS= heat stress, and GAHS = 2 mg L − 1 GSH-Au NCs + heat stress. Data are means ± SE from n  = 3 biological replicates. Different letters indicate significant differences (Duncan’s test, P  < 0.05) The crucial carbon-fixing enzyme Rubisco showed 38.3% reduced activity under heat stress, creating a major constraint in photosynthetic efficiency. However, GSH-Au NCs significantly alleviated heat-induced inhibition of Rubisco, restoring activity by 26.4% compared to stressed plants (Fig.  3 J). This enhancement directly correlated with improved carbon assimilation rates, as evidenced by gas exchange. Chlorophyll fluorescence parameters, which reflect the functional status of photosystem II (PSII), were also severely compromised by heat stress. Fv/Fm, Y(II), qP, and ETR were reduced by 25.8%, 58.2%, 56.3%, and 55.7%, respectively, indicating impaired light energy conversion (Fig.  3 A-E). GSH-Au NCs treatment countered these negative effects, enhancing Fv/Fm by 16.0%, Y(II) by 93.6%, qP by 71.3%, and ETR by 48.2% (Fig.  3 A-E), thereby improving all photosynthetic efficiency under stress. Gas exchange measurements further supported these findings. Heat stress led to substantial reductions in Pn (57.1%), Gs (87.7%), Ci (47.0%), and Tr (66.1%) compared to control plants. However, GSH-Au NC-treated plants exhibited significant recovery, with Pn increasing by 86.1%, GS by 389.0%, Ci by 47.3%, and Tr by 124.3% (Fig.  3 F–I). These results revealed that GSH-Au NCs not only preserve photosynthetic machinery under thermal stress but also enhance stomatal functions and carbon assimilation. Stomatal parameters Heat stress severely affected stomatal characteristics in sweet potato leaves, reducing density (Fig.  4 A) and dimensions, including length (61.1%), width (75.3%), and pore length (61.0%). GSH-Au NCs treatment alleviated these morphological changes, increasing stomatal length by 90.7%, width by 161.4%, and pore length by 60.9% compared to heat-stressed plants (Fig.  4 B-E). GSH-Au NCs promoted stomatal opening and increased stomatal density, suggesting improved gas exchange capacity under stress conditions. Fig. 4 Stomatal characteristics in vegetable sweet potato under normal and heat stress conditions. A Stomatal density under 250 × magnification and scale bar of 200 μm, B stomata under 3000 × magnification and scale bar of 10 μm, C stomatal length, D stomatal width, and ( E ) stomatal pore length under normal and heat stress conditions. Treatments: CK= control, GA = 2 mg L − 1 GSH-Au NCs, HS= heat stress, and GAHS = 2 mg L − 1 GSH-Au NCs + heat stress. Data are means ± SE from n  = 3 biological replicates. Different letters indicate significant differences (Duncan’s test, P  < 0.05) Chloroplast ultrastructure analysis TEM revealed distinct structural differences in sweet potato mesophyll cells under normal and heat-stressed conditions. In normal conditions, both control and GSH-Au NCs-treated mesophyll cells maintained uniform shapes, with chloroplasts exhibiting oval structures and intact thylakoid membranes. Stroma appeared clearly defined, cell walls remained thin with sharp edges, and grana lamellae were tightly stacked with well-organized grana and stroma lamellae. Mitochondria maintained their structural integrity, indicating normal respiration (Fig.  5 ). Fig. 5 Ultrastructural changes in sweet potato vegetable under normal and heat stress conditions. CK= control, GA = GSH-Au NCs, HS= heat stress, GAHS= heat stress + GSH-Au NCs. Abbreviations: CW= Cell Wall, Chl= Chloroplast, M= Mitochondria, C= Cytoplasm, S= Starch, SL= Stroma Lamellae, G= Grana, PG= Plastoglobules. Mesophyll cell has 4000 × magnification and scale bar of 2 μm. Chloroplast structure has 15,000 × magnification and scale bar of 1 μm Heat stress induced significant ultrastructural changes; mesophyll cells became swollen, chloroplasts exhibited abnormal swelling, disrupted thylakoids and irregular stroma, and increased autophagic vesicles suggested intense self-digestion. Cell wall development was impaired, showing blurred edges, while grana lamellae became separated and mitochondria appeared damaged (Fig.  5 ). In contrast, GSH-Au NCs-treated plants under heat stress showed partial restoration of cellular structure. Cell sizes approached control levels, chloroplasts exhibited reduced swelling with partially restored thylakoid structure, and autophagic activity decreased. Cell wall edges regained clarity, grana and stroma lamellae showed improved organization, and mitochondrial swelling was less prominent (Fig.  5 ). These changes highlight the protective role of GSH-Au NCs in maintaining chloroplast ultrastructure under heat stress conditions. Oxidative damage, osmolytes, and non-enzymatic antioxidants Under heat stress, sweet potato leaves exhibited significant oxidative damage, as seen by elevated levels of ROS such as H 2 O 2 and O 2 •− , along with increased lipid peroxidation (MDA) and EL. Histochemical staining revealed distinct visual markers, such as brownish spots for H 2 O 2 , blue spots for O 2 •− , and magenta coloration of MDA, demonstrating their accumulation under heat stress. However, treatment with GSH-Au NCs effectively mitigated these effects by reducing the intensity and number of spots (Fig.  6 A). Concentration determination also showed that heat-stressed plants have escalated the content of H 2 O 2 by 276.7%, O 2 •− by 143.1%, MDA by 481.5%, and EL by 85.7% compared to the control plants ( P  < 0.05; Fig.  6 B-E). Notably, GSH-Au NCs application under heat stress significantly mitigated oxidative damages, as evidenced by a substantial reduction in H 2 O 2 by 37.8%, O 2 •− by 36.6%, MDA by 51.7%, and EL by 26.8% compared to heat-stressed plants (Fig.  6 B-E). Fig. 6 Oxidative damage, osmolytes, and non-enzymatic antioxidants in vegetable sweet potato under normal and heat stress conditions. A Histochemical localization. B - E H 2 O 2 , O 2 •− , MDA, and electrolyte leakage (EL), F proline, G soluble sugars, H total polyphenols, and I ) total flavonoids under normal and heat stress conditions. Treatments: CK= control, GA = 2 mg L − 1 GSH-Au NCs, HS= heat stress, and GAHS = 2 mg L − 1 GSH-Au NCs + heat stress. Data are means ± SE from n  = 3 biological replicates. Different letters indicate significant differences (Duncan’s test, P  < 0.05) To overcome oxidative stress, sweet potato plants accumulate osmolytes (proline and soluble sugars) and non-enzymatic antioxidants (polyphenols and flavonoids). Heat stress triggered a significant increase, with proline rising by 157.4%, soluble sugars by 192.1%, polyphenols by 167.8%, and flavonoids by 239.4% compared to control plants (Fig.  6 F-I). GSH-Au NCs further enhanced this adaptive response by significantly boosting proline (51.7%), polyphenols (38.3%), and flavonoids (62.9%) compared to heat-stressed plants. Interestingly, GSH-Au NCs reduced soluble sugars by 20.1% under heat stress (Fig.  6 F-I). Ascorbate, glutathione and their ratios This study observed significant changes in AsA-GSH cycle in sweet potato plants under heat stress. Specifically, heat stress led to a marked increase in AsA levels by 57.3%, while DHA content decreased by 11.9%, resulting in a significant enhancement of the AsA/DHA ratio by 78.1% compared to the control conditions. When GSH-Au NCs were applied under heat stress, the AsA content increased further by 17.9% and DHA levels declined by an additional 11.2%. This contributed to a 33.8% rise in the AsA/DHA ratio (Fig.  7 A-C). Fig. 7 Content of ascorbate, glutathione, and their ratios. A AsA, B DHA, C AsA/DHA ratio, D GSH, E GSSG, and ( F ) GSH/GSSG ratio under normal and heat stress conditions. Treatments: CK= control, GA = 2 mg L − 1 GSH-Au NCs, HS= heat stress, and GAHS = 2 mg L − 1 GSH-Au NCs + heat stress. Data are means ± SE from n  = 3 biological replicates. Different letters indicate significant differences (Duncan’s test, P  < 0.05) Likewise, the levels of GSH, GSSG, and their corresponding GSH/GSSG ratio were also significantly elevated in response to heat stress by 150.6%, 76.0%, and 43.0%, respectively, relative to control plants. The application of GSH-Au NCs under heat stress further amplified these effects, increasing GSH by 56.0%, GSSG by 20.9%, and the GSH/GSSG ratio by 29.1% (Fig.  7 D-F). Antioxidant enzymes (ASA-GSH cycle) Exposure to heat stress significantly heightened the activities of several key enzymes in sweet potato. SOD, CAT, APX, GR, DHAR, and MDHAR activities increased by 89.2%, 63.6%, 414.4%, 216.7%, 237.1%, and 126.8%, respectively, compared to control plants. The application of GSH-Au NCs under heat stress further elevated the activities of all these mentioned enzymes except CAT. Specifically, GSH-Au NCs treatment increased SOD by 30.1%, APX by 33.6%, GR by 20.9%, DHAR by 54.9%, and MDHAR by 46.6%. Surprisingly, CAT activity decreased by 20.5% following GSH-Au NCs treatment (Fig.  8 A-F). Fig. 8 Antioxidant enzyme activities under normal and heat stress conditions. A - F GSH-AsA cycle enzymes (SOD, CAT, APX, GR, DHAR, and MDHAR). G-H) POD and GST activities. Treatments: CK= control, GA = 2 mg L − 1 GSH-Au NCs, HS= heat stress, and GAHS = 2 mg L − 1 GSH-Au NCs + heat stress. Data are means ± SE from n  = 3 biological replicates. Different letters indicate significant differences (Duncan’s test, P  < 0.05) POD and GST play crucial roles in scavenging H 2 O 2 and detoxifying lipid peroxides, respectively. Heat stress elevated POD and GST activities by 197.0% and 83.8%, respectively. However, GSH-Au NCs treatment further increased POD by 33.5% and GST by 53.6% under heat stress conditions (Fig. G-H). Transcriptional expression of genes Under heat stress conditions, the transcriptional levels of multiple antioxidant enzymes–related genes were significantly upregulated in sweet potato plants, with the exception of CAT , which showed downregulation. Notably, SOD , APX , GR , DHAR , and MDHAR showed pronounced rises in transcriptional activity (Fig.  9 A-F). Treatments with GSH-Au NCs further enhanced the expression of these upregulated genes in heat-stressed plants, while CAT expression remained suppressed (Fig.  9 A-F), consistent with the observed enzymatic activity pattern. Similarly, POD and GST expression levels increased under heat stress. However, GSH-Au NCs treatment resulted in a non-significant reduction in POD levels, while GST expression was further enhanced under heat stress conditions (Fig.  9 G). Fig. 9 Transcriptional expression of genes encoding antioxidant enzymes under heat stress conditions. A - F Genes encoding GSH-AsA cycle enzymes ( SOD , CAT , APX , GR , MDHAR , and DHAR ). G , H

POD and GST genes. Treatments: CK= control, HS= heat stress, and GAHS = 2 mg L − 1 GSH-Au NCs + heat stress. Data are means ± SE from n  = 3 biological replicates. Different letters indicate significant differences (Duncan’s test, P  < 0.05) Methylglyoxal and glyoxalase enzyme activities Heat stress significantly increased the accumulation of MG in sweet potato plants, with MG levels rising by 137.5% compared to control plants. However, application of GSH-Au NCs under heat stress effectively reduced MG content by 29.3%. Conversely, heat stress suppressed the activities of Gly I and Gly II by 37.7% and 40.3%, respectively, relative to control plants. Application of GSH-Au NCs under heat stress conditions significantly restored their activities, with Gly I and Gly II increasing by 36.9% and 35.0%, respectively (Fig.  10 A-C). Fig. 10 Methylglyoxal and glyoxalase enzymes under normal and heat stress conditions. A Methylglyoxal (MG) content, B Glyoxalase I (Gly I), and ( C ) Gly II activity. Treatments: CK= control, GA = 2 mg L − 1 GSH-Au NCs, HS= heat stress, and GAHS = 2 mg L − 1 GSH-Au NCs + heat stress. Data are means ± SE from n  = 3 biological replicates. Different letters indicate significant differences (Duncan’s test, P  < 0.05) Pearson correlation analysis The Pearson correlation matrix displayed significant relationships among key physiological parameters in heat-stressed sweet potato plants. The results showed strong positive correlations between oxidative stress markers (MDA, H 2 O 2 , O 2 •− , and MG), EL, and soluble sugar accumulation under heat stress conditions. In contrast, GSH-Au NCs treatment revealed significant negative correlations with these stress indicators (Fig.  11 ; Table S3), suggesting its protective role against oxidative damage. Furthermore, GSH-Au NCs exhibited positive correlations with growth and physiological parameters, such as plant biomass, gas exchange parameters, chlorophyll and carotenoid content, chlorophyll fluorescence, Rubisco activity, and components of the antioxidant defense system (except CAT). Additionally, positive associations were observed between GSH-Au NCs and glyoxalase enzymes, secondary metabolites accumulation, proline content, and stomatal morphological traits (Fig.  11 ; Table S3). Together, these results highlight the efficacy of GSH-Au NCs in enhancing the antioxidant defense mechanisms and improving thermotolerance in sweet potato plants. Fig. 11 Pearson correlation analysis of growth, physio-biochemical parameters in sweet potato plants under heat stress with GSH-Au NCs treatments. The abbreviations of given parameters are the same as mentioned above; SFW (shoot fresh weight), SDW (shoot dry weight), RFW (root fresh weight), RWC (relative water content), and so on. In the correlation matrix, orange-colored lines indicate positive correlation, while light blue dotted lines indicate negative correlation Discussion Heat stress impaired sweet potato growth, biomass, RWC, and root morphology (Fig. 1 ; S2), likely due to disrupted cell division, metabolism, and photosynthesis. These changes hinder nutrient and water uptake, further intensifying stress, as observed in various plants under stress conditions [ 10 , 40 , 41 ]. Unlike intensive conventional agriculture, nanotechnology enables sustainable production using nanomaterials that may simultaneously support stress adaptation and nutrient uptake efficiency [ 15 , 42 ]. In the present study, foliar application of GSH-Au NCs (2 mg L − 1 ) was associated with substantial mitigation of heat stress effects, contributing to recovery in shoot and root biomass, plant height, leaf development, and root architecture in sweet potato plants (Fig. 1 ; S2). These observations are consistent with studies where nanomaterials (Se, Ag, and TiO 2 ) improved growth, biomass, and RWC in crops such as tomato, wheat, and Chrysanthemum morifolium under heat stress [ 43 – 46 ]. The observed recovery in root traits (Fig. S2) may have helped compensate for stress-induced photoassimilate imbalances, potentially supporting nutrient and water uptake, promoting plant growth through enhanced turgor and cell expansion [ 47 , 48 ]. GSH-Au NCs were also associated with elevated RWC, likely linked to osmolyte accumulation, particularly proline. This pattern is consistent with reports on carbon-NPs. AgNPs and ZnONPs, which contributed to improved cellular homeostasis, membrane integrity, and water retention under heat stress conditions [ 45 , 49 , 50 ]. These responses suggest that GSH-Au NCs may support stress mitigation across growth, water balance, photosynthetic performance, metabolism, and structural integrity. Photosynthesis is highly sensitive to elevated temperatures, which can impair biochemical and structural components of the photosynthesis apparatus [ 51 ]. In this study, heat stress drastically reduced gas exchange parameters (Fig. 3 F-I), likely limiting CO 2 uptake and evaporative cooling, as previously reported in tomato, sorghum, and Chrysanthemum morifolium [ 52 , 53 ]. Heat stress also suppressed chlorophyll fluorescence (Fig. 3 A-E), indicating possible photosystem II (PSII) damage and impaired electron transport, contributing to reduced Fv/Fm ratio and impeding antenna molecules. These changes may result from altered enzyme activity, guard cell dysfunction, and ETC inhibition [ 47 ]. Accordingly, reduced Rubisco activity, chlorophyll and carotenoid content, and intensity were observed in sweet potato leaves (Figs. 2 and 3 ). However, GSH-Au NCs was associated with improved Gs, Tr, Pn, and Ci, suggesting enhanced mesophyll conductance and carbon assimilation. GSH-Au NCs treatment also contributed to recovery in chlorophyll fluorescence parameters (Fv/Fm, Y(II), qP, and ETR), increased chlorophyll and carotenoid content, and pigment fluorescence intensity (Figs. 2 and 3 ), and reduced qN, indicating improved PSII efficiency and photoprotective capacity. These responses are consistent with reports on Au, TiO 2 , MnO, and Se nanomaterials, which have been associated with mitigation of photoinhibition, ROS scavenging and preservation of gas exchange in tomato, coriander, and wheat [ 47 , 54 – 57 ]. Enhanced Rubisco activity (Fig. 2 J) further supports improved regulation of the Calvin-Benson cycle, consistent with previous findings on TiO 2 and CeO 2 [ 58 , 59 ]. These improvements are consistent with reduced oxidative stress contributing to sustained photosynthetic performance and energy production in sweet potato under heat stress. Heat stress can damage stomata and ultrastructure, disrupting gas exchange and photosynthetic efficiency [ 10 ]. In this study, sweet potato leaves exhibited reduced stomatal density, length, width, and pore length (Fig. 4 ), along with disorganized mesophyll cells, chloroplasts, and impaired mitochondria (Fig. 5 ). These structural alterations may hinder light-dependent reactions and mitochondrial ATP synthesis [ 8 , 52 ]. GSH-Au NCs was associated with improved stomatal traits (density, length, width, and pore length) (Fig. 4 ), potentially facilitating gas exchange, evaporative cooling, and CO 2 uptake. These observations align with reports on CeO 2 , ZnO, and Zn-Si NPs enhancing stomatal opening in sorghum and pea [ 52 , 60 ]. Electron microscopy revealed that GSH-Au NCs were associated with preserved mesophyll integrity, chloroplast structure (thylakoids, stroma, and grana), and mitochondrial morphology (Fig. 5 ), likely reducing oxidative damage. Preserved chloroplasts may support photosynthesis, while intact mitochondria may ensure energy metabolism. GSH-Au NC treatment also appeared to increase plastoglobuli accumulation, which may contribute to lipid metabolism and stress resilience. These structural responses are consistent with previous studies on AuNPs and SiO 2 NPs in wheat and rice, and CeO 2 NPs under cobalt stress [ 57 , 61 , 62 ]. These findings suggest that GSH-Au NCs may contribute to maintaining stomatal function and ultrastructure, thereby supporting photosynthetic recovery. Heat stress triggers excessive ROS generation, leading to oxidative and carbonyl stress that can damage lipids, proteins, and DNA [ 8 , 11 ]. In this study, heat-stressed sweet potato plants showed elevated H 2 O 2 , O 2 •− , MDA, MG, and EL, confirmed by histochemical staining (H 2 O 2 , O 2 •− , and MDA; Fig. 6 A-E). These effects are likely linked to photosynthesis decline and excess electron leakage. Soluble sugar accumulation (Fig. 6 G) may reflect osmotic adjustment but could also indicate disrupted carbohydrate metabolism [ 63 ]. GSH-Au NCs treatment was associated with reduced oxidative markers (H 2 O 2 , O 2 •− , MDA, MG, and EL) and less intense histochemical staining (Fig. 6 A-E). These findings are consistent with previous studies on Se, S, CeO 2 , ZnO, and TiO 2 nanomaterials suppressing oxidative damage under abiotic stress [ 44 , 47 , 64 ]. GSH-Au NCs also appeared to reduce soluble sugar levels, suggesting restored photosynthetic activity and carbohydrate metabolism toward growth and energy production [ 63 , 65 ]. Furthermore, GSH-Au NCs were associated with increased glyoxalase I and II activity (Fig. 10 ), likely contributing to MG detoxification through the GSH-dependent glyoxalase pathway. This pattern aligns with previous reports on nanoparticle-enhanced glyoxalase activity in other plant systems [ 47 , 66 ]. Together, these coordinated responses suggest that GSH-Au NCs may strengthen redox homeostasis and thermotolerance through multiple antioxidant layers. The AsA-GSH cycle plays a central role in antioxidant defense by detoxifying ROS and maintaining redox balance under heat stress [ 67 ]. Our results show that GSH-Au NCs was associated with increased AsA and GSH levels, elevated AsA/DHA and GSH/GSSG ratios (Fig. 7 A-F), indicating enhanced antioxidant capacity. Treatment also upregulated both the enzymatic activity (SOD, APX, GR, DHAR, and MDHAR) and gene expression of AsA-GSH cycle (Figs. 8 and 9 ). These responses are consistent with reports on carbon nanotubes, Se, S, and MnO nanomaterials enhancing AsA-GSH components in in Paeonia ostii , wheat, and tomato [ 47 , 66 , 68 , 69 ]. Upregulation of these AsA-GSH cycle genes may involve HSF, MYB, and WRKY transcription factors, potentially activated by ROS or NP-induced MAPK signaling [ 50 , 70 ]. Interestingly, GSH-Au NCs were associated with reduced CAT activity (Figs. 8 B and 9 B), likely due to decreased H 2 O 2 load from enhanced AsA-GSH pathway activity, consistent with ZnO-NP studies [ 71 , 72 ]. In contrast, increased POD and GST activity (Fig. 8 G-H) may aid in peroxides and xenobiotics detoxification [ 63 , 66 ]. GSH-Au NCs was also associated with enhanced proline levels (Fig. 6 F), likely supporting osmotic balance, membrane stability, and ROS scavenging in sweet potato plants under heat stress. This is consistent with reports on various nanoparticles increasing proline accumulation under abiotic stress [ 54 , 70 ]. Additionally, GSH-Au NC treatment contributed to higher secondary metabolites (polyphenols and flavonoids) in sweet potato leaves under heat stress (Fig. 6 H-I), which may contribute to ROS scavenging and photosynthetic recovery through phenylpropanoid pathway [ 13 , 73 ]. These observations align with effects of FeNPs, SeNPs, Ag NPs, and SiNPs in various plants [ 49 , 63 , 66 , 74 ]. Overall, the present study demonstrated that GSH-Au NCs (2 mg L − 1 ) was associated with substantial alleviation of heat stress in sweet potato plants. Mechanistically, GSH-Au NCs likely modulates redox balance by direct ROS scavenging (evidenced by reduced H 2 O 2 , O 2 •− , MDA, and EL) and transcriptional upregulation of AsA-GSH genes ( SOD , APX , GR , MDHAR , and DHAR ), consistent with Au NP effects [ 25 – 27 ]. Speculatively, Au’s catalytic properties may enhance GSH stability and regeneration, though enzyme kinetics require further confirmation. Similar to Au NPs in wheat (enhanced ROS scavenging) [ 26 ], our results show reduced oxidative damage; however, the GSH-Au NC synergy uniquely elevated GSH/GSSG by 29.1%, a response not reported in separate applications, highlighting novelty while aligning with GSH’s role in tomato [ 23 ]. Additionally, increased glyoxalase activities (Gly I and Gly II) by GSH-Au NC treatment contributed to lower methylglyoxal levels, further supporting redox homeostasis. These coordinated responses collectively contributed to recovery in growth, biomass, photosynthetic efficiency, and cellular ultrastructure while minimizing oxidative damage through ROS scavenging and AsA-GSH cycle activation. These findings position GSH-Au NCs as a promising, sustainable solution for crop resilience under abiotic stress. However, limitations include the lack of direct confirmation of nanocluster uptake and distribution via ICP-MS or TEM, reliance on short-term greenhouse assessments that may not fully reflect field variability or long-term outcomes, and absence of direct toxicity profiling. Future studies should prioritize localization, extended field trials, and multi-season evaluations. Practically, GSH-Au NCs offers a low-cost and eco-friendly solution for high temperature regions, but these limitations necessitate cautious interpretation. Conclusions This study demonstrates the first comprehensive evidence that GSH-Au NCs (2 mg L − 1 ) was associated with substantial enhancement of thermotolerance in vegetable sweet potato, contributing to improved growth, biomass, photosynthetic efficiency, and structural integrity. These effects were linked to reduced oxidative and carbonyl stress through activation of the AsA-GSH cycle, upregulation of antioxidant enzyme activities and gene expression (SOD, APX, GR, DHAR, MDHAR, POD, and GST), enhanced glyoxalase system (Gly I and II), and osmolyte (proline) accumulation. GSH-Au NC treatment also contributed to recovery in stomatal function, chloroplast and mitochondria ultrastructure, and increased secondary metabolites (polyphenols and flavonoids), further supporting resilience. These findings highlight GSH-Au NCs as a sustainable, cost-effective, and eco-friendly strategy for mitigating heat stress in sweet potato and potentially other crops, advancing beyond prior work on individual GSH supplementation or Au nanomaterials by demonstrating their combined nanocluster synergy. However, limitations include lack of direct However, further research is needed to confirm direct uptake, elucidate molecular mechanisms, validate these findings under field conditions, and assess long-term environmental impact and safety of nanomaterials application in agricultural crop systems. Supplementary Information

Supplementary Material 1: Table S1. Impact of glutathione-gold nanoclusters (GSH-Au NCs) on the growth and biomass of vegetable sweet potato under normal and heat stress conditions. Table S2. List of the primers of genes encoding GSH-AsA cycle enzymes and antioxidant POD and GST enzymes. Table S3. Table of Pearson’s correlation analysis. Fig. S1. Characterization of GSH-Au nanoclusters (GSH-Au NCs). Fig. S2. Impact of glutathione-gold nanoclusters (GSH-Au NCs) on the root traits of vegetable sweet potato under normal and heat stress conditions.

Acknowledgements This research was supported by the Project of Sanya Yazhou Bay Science and Technology City “Yazhou Bay” Jingying Talent Project (SKJC-JYRC-2024-14; SKJC-JYRC-2024-25), the Scientific Research Start-up Funding Project of Hainan University (XJ2400007871), the earmarked fund for CARS-10-Sweetpotato, and the Innovation Platform for Academicians of Hainan Province (YSPTZX202206). We are also highly thankful to the National Tropical Plants Germplasm Resource Center. Authors’ contributions Sunjeet Kumar: Conceptualization, Project administration, Resources, Writing - original draft, Validation, Methods, Visualization. Gurmendar: Investigation, Formal analysis, Writing original draft. Rui Yu: Investigation. Muhammad Ikram: Software. Jingjing Kou: Writing review \& editing. Muhammad Abbas Khan: Writing - review \& editing. Mengzhao Wang: Visualization, Project administration, Data curation, Validation. Guopeng Zhu: Conceptualization, Resources, Supervision, Funding acquisition. Data availability Data will be made available on request. Declarations Ethics approval and consent to participate Not applicable. Consent for publication Not applicable. Competing interests The authors declare no competing interests. Footnotes Publisher’s note Springer Nature remains neutral with regard to jurisdictional claims in published maps and institutional affiliations. Sunjeet Kumar and Gurmendar contributed equally to this work. Contributor Information Mengzhao Wang, Email: mzwang@hainanu.edu.cn. Guopeng Zhu, Email: zhuguopeng@hainanu.edu.cn. References 1. Shelake RM, Kadam US, Kumar R, Pramanik D, Singh AK, Kim J-Y. Engineering drought and salinity tolerance traits in crops through CRISPR-mediated genome editing: Targets, tools, challenges, and perspectives. Plant Commun. 2022;3:100417. 10.1016/j.xplc.2022.100417.

2. Teng Z, Chen C, He Y, Pan S, Liu D, Zhu L, et al. Melatonin confers thermotolerance and antioxidant capacity in Chinese cabbage. Plant Physiol Biochem. 2024;212:108736. 10.1016/j.plaphy.2024.108736.

3. Tan Y, Cao Y, Mou F, Liu B, Wu H, Zou S, et al. Transcriptome profiling of two Camellia japonica cultivars with different heat tolerance reveals heat stress response mechanisms. Plants. 2024;13:3089. 10.3390/plants13213089.

4. El-Ramady H, Prokisch J, El-Mahrouk ME, Bayoumi YA, Shalaby TA, Brevik EC, et al. Nano-food farming approaches to mitigate heat stress under ongoing climate change: A review. Agriculture. 2024;14:656. 10.3390/agriculture14050656. 5. Kumar A, Bhattacharya T, Mukherjee S, Sarkar B. A perspective on biochar for repairing damages in the soil–plant system caused by climate change-driven extreme weather events. Biochar. 2022;4:22. 10.1007/s42773-022-00148-z. 6. Guo S, Hu X, Yu F, Mu L. Heat waves coupled with nanoparticles induce yield and nutritional losses in rice by regulating stomatal closure. ACS Nano. 2024;18:14276–89. 10.1021/acsnano.3c13165.

7. Lee C-C, Zeng M, Luo K. How does climate change affect food security? Evidence from China. Environ Impact Assess Rev. 2024;104:107324. 10.1016/j.eiar.2023.107324. 8. Zhang L, Chang Q, Hou X, Wang J, Chen S, Zhang Q, et al. The effect of high-temperature stress on the physiological indexes, chloroplast ultrastructure, and photosystems of two herbaceous peony cultivars. J Plant Growth Regul. 2023;42:1631–46. 10.1007/s00344-022-10647-9. 9. Toprak S, Coşkun ÖF. Heat stress mitigation by zinc oxide nanoparticles in pepper and watermelon. BMC Agric. 2026;2:2. 10.1186/s44399-025-00024-8. 10. Hu D, Zhang X, Xue P, Nie Y, Liu J, Li Y, et al. Exogenous melatonin ameliorates heat damages by regulating growth, photosynthetic efficiency and leaf ultrastructure of carnation. Plant Physiol Biochem. 2023;198:107698. 10.1016/j.plaphy.2023.107698.

11. Li Z-G, Xu Y, Bai L-K, Zhang S-Y, Wang Y. Melatonin enhances thermotolerance of maize seedlings ( Zea mays L.) by modulating antioxidant defense, methylglyoxal detoxification, and osmoregulation systems. Protoplasma. 2019;256:471–90. 10.1007/s00709-018-1311-4.

12. Sun C, Meng S, Wang B, Zhao S, Liu Y, Qi M, et al. Exogenous melatonin enhances tomato heat resistance by regulating photosynthetic electron flux and maintaining ROS homeostasis. Plant Physiol Biochem. 2023;196:197–209. 10.1016/j.plaphy.2023.01.043.

13. Kumar S, Yu R, Liu Y, Liu Y, Khan MN, Liu Y, et al. Exogenous melatonin enhances heat stress tolerance in sweetpotato by modulating antioxidant defense system, osmotic homeostasis and stomatal traits. Hortic Plant J. 2025;11:431–45. 10.1016/j.hpj.2023.12.006. 14. Chen F, Shen Z, Shi R, Zhang X, Zhang H, Li W, et al. Carbon dots-mediated plant adaptive responses to abiotic stress. Mater Res Bull. 2025;182. 10.1016/j.materresbull.2024.113137. October 2024:113137. 15. Ahmed T, Masood HA, Noman M, AL-Huqail AA, Alghanem SM, Khan MM, et al. Biogenic silicon nanoparticles mitigate cadmium (Cd) toxicity in rapeseed ( Brassica napus L.) by modulating the cellular oxidative stress metabolism and reducing Cd translocation. J Hazard Mater. 2023;459:132070. 10.1016/j.jhazmat.2023.132070.

16. Ghorbani A, Emamverdian A, Pehlivan N, Zargar M, Razavi SM, Chen M. Nano-enabled agrochemicals: mitigating heavy metal toxicity and enhancing crop adaptability for sustainable crop production. J Nanobiotechnol. 2024;22:91. 10.1186/s12951-024-02371-1. 17. Farooq MA, Islam F, Ayyaz A, Chen W, Noor Y, Hu W, et al. Mitigation effects of exogenous melatonin-selenium nanoparticles on arsenic-induced stress in Brassica napus. Environ Pollut. 2022;292:118473. 10.1016/j.envpol.2021.118473.

18. Khan MN, Li Y, Khan Z, Chen L, Liu J, Hu J, et al. Nanoceria seed priming enhanced salt tolerance in rapeseed through modulating ROS homeostasis and α-amylase activities. J Nanobiotechnol. 2021;19:276. 10.1186/s12951-021-01026-9. 19. Ulhassan Z, Yang S, He D, Khan AR, Salam A, Azhar W, et al. Seed priming with nano-silica effectively ameliorates chromium toxicity in Brassica napus . J Hazard Mater. 2023;458:131906. 10.1016/j.jhazmat.2023.131906.

20. Wang C, Liu X, Li J, Yue L, Yang H, Zou H, et al. Copper nanoclusters promote tomato ( Solanum lycopersicum L.) yield and quality through improving photosynthesis and roots growth. Environ Pollut. 2021;289:117912. 10.1016/j.envpol.2021.117912.

21. Gao M, Chang J, Wang Z, Zhang H, Wang T. Advances in transport and toxicity of nanoparticles in plants. J Nanobiotechnol. 2023;21:75. 10.1186/s12951-023-01830-5. 22. Mahakham W, Sarmah AK, Maensiri S, Theerakulpisut P. Nanopriming technology for enhancing germination and starch metabolism of aged rice seeds using phytosynthesized silver nanoparticles. Sci Rep. 2017;7:1–21. 10.1038/s41598-017-08669-5.

23. Nahar K, Hasanuzzaman M, Alam MM, Fujita M. Exogenous glutathione confers high temperature stress tolerance in mung bean ( Vigna radiata L.) by modulating antioxidant defense and methylglyoxal detoxification system. Environ Exp Bot. 2015;112:44–54. 10.1016/j.envexpbot.2014.12.001. 24. Kumar S, Wang S, Wang M, Zeb S, Khan MN, Chen Y, et al. Enhancement of sweetpotato tolerance to chromium stress through melatonin and glutathione: Insights into photosynthetic efficiency, oxidative defense, and growth parameters. Plant Physiol Biochem. 2024;208:108509. 10.1016/j.plaphy.2024.108509.

25. Feichtmeier NS, Walther P, Leopold K. Uptake, effects, and regeneration of barley plants exposed to gold nanoparticles. Environ Sci Pollut Res. 2015;22:8549–58. 10.1007/s11356-014-4015-0. 26. Ferrari E, Barbero F, Busquets-Fité M, Franz-Wachtel M, Köhler H-R, Puntes V, et al. Growth-promoting gold nanoparticles decrease stress responses in Arabidopsis seedlings. Nanomaterials. 2021;11:3161. 10.3390/nano11123161.

27. Venzhik Y, Deryabin A, Naraikina N, Zhukova K, Dykman L. The influence of Au-based nanoparticles on some physiological, biochemical and molecular characteristics of wheat plants during low temperature hardening. Plant Physiol Biochem. 2024;213:108837. 10.1016/j.plaphy.2024.108837.

28. Ji CY, Jin R, Xu Z, Kim HS, Lee C-J, Kang L, et al. Overexpression of Arabidopsis P3B increases heat and low temperature stress tolerance in transgenic sweetpotato. BMC Plant Biol. 2017;17:139. 10.1186/s12870-017-1087-2.

29. Yu J, Su D, Yang D, Dong T, Tang Z, Li H, et al. Chilling and heat stress-induced physiological changes and microRNA-related mechanism in sweetpotato ( Ipomoea batatas L). Front Plant Sci. 2020;11:687. 10.3389/fpls.2020.00687.

30. Kang L, Kim HS, Kwon YS, Ke Q, Ji CY, Park S-C, et al. IbOr regulates photosynthesis under heat stress by stabilizing IbPsbP in sweetpotato. Front Plant Sci. 2017;8:00989. 10.3389/fpls.2017.00989. 31. Wijewardana C, Reddy KR, Shankle MW, Meyers S, Gao W. Low and high-temperature effects on sweetpotato storage root initiation and early transplant establishment. Sci Hortic. 2018;240:38–48. 10.1016/j.scienta.2018.05.052. 32. Kumar S, Liu Y, Wang M, Khan MN, Wang S, Li Y, et al. Alleviating sweetpotato salt tolerance through exogenous glutathione and melatonin: A profound mechanism for active oxygen detoxification and preservation of photosynthetic organs. Chemosphere. 2024;350:141120. 10.1016/j.chemosphere.2024.141120.

33. Mumtaz MA, Hao Y, Mehmood S, Shu H, Zhou Y, Jin W, et al. Physiological and transcriptomic analysis provide molecular insight into 24-epibrassinolide mediated Cr(VI)-toxicity tolerance in pepper plants. Environ Pollut. 2022;306:119375. 10.1016/j.envpol.2022.119375.

34. Khan MA, Kumar S, Wang Q, Wang M, Fahad S, Nizamani MM, et al. Influence of polyvinyl chloride microplastic on chromium uptake and toxicity in sweet potato. Ecotoxicol Environ Saf. 2023;251:114526. 10.1016/j.ecoenv.2023.114526.

35. Altaf MA, Hao Y, Shu H, Mumtaz MA, Cheng S, Alyemeni MN, et al. Melatonin enhanced the heavy metal-stress tolerance of pepper by mitigating the oxidative damage and reducing the heavy metal accumulation. J Hazard Mater. 2023;454:131468. 10.1016/j.jhazmat.2023.131468.

36. Kaya C, Ashraf M, Alyemeni MN, Rinklebe J, Ahmad P. Alleviation of arsenic toxicity in pepper plants by aminolevulinic acid and heme through modulating its sequestration and distribution within cell organelles. Environ Pollut. 2023;330:121747. 10.1016/j.envpol.2023.121747.

37. Mohammadi H, Abdollahi-Bastam S, Aghaee A, Ghorbanpour M. Foliar-applied silicate potassium modulates growth, phytochemical, and physiological traits in Cichorium intybus L. under salinity stress. BMC Plant Biol. 2024;24:288. 10.1186/s12870-024-05015-6.

38. Kumar S, Huang X, Li G, Ji Q, Zhou K, Zhu G, et al. Comparative transcriptomic analysis provides novel insights into the blanched stem of Oenanthe javanica . Plants. 2021;10:2484. 10.3390/plants10112484.

39. Kumar S, Huang X, Ji Q, Qayyum A, Zhou K, Ke W, et al. Influence of blanching on the gene expression profile of phenylpropanoid, flavonoid and vitamin biosynthesis, and their accumulation in Oenanthe javanica . Antioxidants. 2022;11:470. 10.3390/antiox11030470.

40. Ullah I, Toor MD, Yerlikaya BA, Mohamed HI, Yerlikaya S, Basit A, et al. High-temperature stress in strawberry: understanding physiological, biochemical and molecular responses. Planta. 2024;260:118. 10.1007/s00425-024-04544-6.

41. Xing X, Ding Y, Jin J, Song A, Chen S, Chen F, et al. Physiological and transcripts analyses reveal the mechanism by which melatonin alleviates heat stress in chrysanthemum seedlings. Front Plant Sci. 2021;12:673236. 10.3389/fpls.2021.673236.

42. Yao Y, Yue L, Cao X, Chen F, Li J, Cheng B, et al. Carbon dots embedded in nanoporous SiO 2 nanoparticles for enhancing photosynthesis in agricultural crops. ACS Appl Nano Mater. 2023;6:110–8. 10.1021/acsanm.2c03843. 43. El-Saadony MT, Saad AM, Najjar AA, Alzahrani SO, Alkhatib FM, Shafi ME, et al. The use of biological selenium nanoparticles to suppress Triticum aestivum L. crown and root rot diseases induced by Fusarium species and improve yield under drought and heat stress. Saudi J Biol Sci. 2021;28:4461–71. 10.1016/j.sjbs.2021.04.043.

44. El-Saadony MT, Saad AM, Soliman SM, Salem HM, Desoky E-SM, Babalghith AO, et al. Role of nanoparticles in enhancing crop tolerance to abiotic stress: A comprehensive review. Front Plant Sci. 2022;13:946717. 10.3389/fpls.2022.946717.

45. Haghighi M, Abolghasemi R, Teixeira da Silva JA. Low and high temperature stress affect the growth characteristics of tomato in hydroponic culture with Se and nano-Se amendment. Sci Hortic. 2014;178:231–40. 10.1016/j.scienta.2014.09.006. 46. Seliem MK, Hafez Y, El-Ramady H. Using of nano-selenium in reducing the negative effects of high temperature stress on Chrysanthemum morifolium Ramat. J Sustainable Agricultural Sci. 2020;46:47–59. 10.21608/jsas.2020.23905.1203. 47. Babzada SA, Raja V, Bhat AH, Qadir SU, Radhakrishnan A, Kumar N, et al. Alleviating lanthanum stress in tomato plants using MnO nanoparticles and triacontanol: Impacts on growth, photosynthesis, and antioxidant defense. J Hazard Mater. 2025;491:137746. 10.1016/j.jhazmat.2025.137746.

48. Zhang K, Han X, Fu Y, Khan Z, Zhang B, Bi J, et al. Biochar coating promoted rice growth under drought stress through modulating photosynthetic apparatus, chloroplast ultrastructure, stomatal traits and ROS homeostasis. Plant Physiol Biochem. 2024;216. 10.1016/j.plaphy.2024.109145. September:109145. 49. Iqbal M, Raja NI, Mashwani Z, Wattoo FH, Hussain M, Ejaz M, et al. Assessment of AgNPs exposure on physiological and biochemical changes and antioxidative defence system in wheat ( Triticum aestivum L) under heat stress. IET Nanobiotechnol. 2019;13:230–6. 10.1049/iet-nbt.2018.5041.

50. Cao Y, Turk K, Bibi N, Ghafoor A, Ahmed N, Azmat M, et al. Nanoparticles as catalysts of agricultural revolution: Enhancing crop tolerance to abiotic stress: a review. Front Plant Sci. 2025;15:1510482. 10.3389/fpls.2024.1510482.

51. Teng L, Qing L, Shumei W, Xuepeng Z, Yuanquan C, Wangsheng G, et al. High temperature effects on maize photosynthesis during stress and recovery phase at the seed setting stage. BMC Plant Biol. 2025;25:454. 10.1186/s12870-025-06047-2.

52. Djanaguiraman M, Belliraj N, Bossmann SH, Prasad PVV. High-temperature stress alleviation by selenium nanoparticle treatment in grain sorghum. ACS Omega. 2018;3:2479–91. 10.1021/acsomega.7b01934.

53. Jahan MS, Hasan MM, Alotaibi FS, Alabdallah NM, Alharbi BM, Ramadan KMA, et al. Exogenous putrescine increases heat tolerance in tomato seedlings by regulating chlorophyll metabolism and enhancing antioxidant defense efficiency. Plants. 2022;11:1038. 10.3390/plants11081038.

54. Omar AA, Heikal YM, Zayed EM, Shamseldin SAM, Salama YE, Amer KE, et al. Conferring of drought and heat stress tolerance in wheat ( Triticum aestivum L.) genotypes and their response to selenium nanoparticles application. Nanomaterials. 2023;13:998. 10.3390/nano13060998.

55. Qi M, Liu Y, Li T. Nano-TiO2 improve the photosynthesis of tomato leaves under mild heat stress. Biol Trace Elem Res. 2013;156:323–8. 10.1007/s12011-013-9833-2.

56. Sardar R, Ahmed S, Yasin NA. Titanium dioxide nanoparticles mitigate cadmium toxicity in Coriandrum sativum L. through modulating antioxidant system, stress markers and reducing cadmium uptake. Environ Pollut. 2022;292:118373. 10.1016/j.envpol.2021.118373.

57. Venzhik Y, Deryabin A, Popov V, Dykman L, Moshkov I. Priming with gold nanoparticles leads to changes in the photosynthetic apparatus and improves the cold tolerance of wheat. Plant Physiol Biochem. 2022;190:145–55. 10.1016/j.plaphy.2022.09.006.

58. Cao Z, Stowers C, Rossi L, Zhang W, Lombardini L, Ma X. Physiological effects of cerium oxide nanoparticles on the photosynthesis and water use efficiency of soybean ( Glycine max (L.) Merr). Environ Sci Nano. 2017;4:1086–94. 10.1039/C7EN00015D. 59. Sidhu AK, Sharma M, Bhickchand Agrawal S, Pradip Bhavsar P, Samota MK. Nanomaterial strategies for enhancing plant resilience in the face of temperature stress. CABI Agric Bioscience. 2024;5:60. 10.1186/s43170-024-00255-w. 60. Elshoky HA, Yotsova E, Farghali MA, Farroh KY, El-Sayed K, Elzorkany HE, et al. Impact of foliar spray of zinc oxide nanoparticles on the photosynthesis of Pisum sativum L. under salt stress. Plant Physiol Biochem. 2021;167:607–18. 10.1016/j.plaphy.2021.08.039.

61. Muhammad S, Ulhassan Z, Munir R, Yasin MU, Islam F, Zhang K, et al. Nanosilica and salicylic acid synergistically regulate cadmium toxicity in rice. Environ Pollut. 2025;364:125331. 10.1016/j.envpol.2024.125331.

62. Salam A, Qi J, Fan X, Khan AR, Kah M, Zeeshan M, et al. Cerium oxide nanoparticle protects maize from cobalt stress: Insights from transcriptomics and oxidative stress response analysis. ACS Appl Mater Interfaces. 2025;17:36455–68. 10.1021/acsami.5c05835.

63. Shahzad R, Koerniati S, Harlina PW, Hastilestari BR, Djalovic I, Prasad PVV. Iron oxide nanoparticles enhance alkaline stress resilience in bell pepper by modulating photosynthetic capacity, membrane integrity, carbohydrate metabolism, and cellular antioxidant defense. BMC Plant Biol. 2025;25:170. 10.1186/s12870-025-06180-y.

64. Rehman A, Khan S, Sun F, Peng Z, Feng K, Wang N, et al. Exploring the nano-wonders: Unveiling the role of nanoparticles in enhancing salinity and drought tolerance in plants. Front Plant Sci. 2024;14:1324176. 10.3389/fpls.2023.1324176.

65. Dang K, Mu J, Tian H, Gao D, Zhou H, Guo L, et al. Zinc regulation of chlorophyll fluorescence and carbohydrate metabolism in saline-sodic stressed rice seedlings. BMC Plant Biol. 2024;24:464. 10.1186/s12870-024-05170-w.

66. Shah T, Khan Z, Alahmadi TA, Shah MA, Ahmad MZ, Rasool S, et al. Nanoselenium inhibits chromium toxicity in wheat plants by modifying the antioxidant defense system, ascorbate glutathione cycle, and glyoxalase system. Environ Exp Bot. 2024;220:105697. 10.1016/j.envexpbot.2024.105697. 67. Jahan MS, Shu S, Wang Y, Chen Z, He M, Tao M, et al. Melatonin alleviates heat-induced damage of tomato seedlings by balancing redox homeostasis and modulating polyamine and nitric oxide biosynthesis. BMC Plant Biol. 2019;19:414. 10.1186/s12870-019-1992-7.

68. Riaz M, Zhao S, Kamran M, Ur Rehman N, Mora-Poblete F, Maldonado C, et al. Effect of nano-silicon on the regulation of ascorbate-glutathione contents, antioxidant defense system and growth of copper stressed wheat ( Triticum aestivum L.) seedlings. Front Plant Sci. 2022;13:986991. 10.3389/fpls.2022.986991.

69. Zhao D, Wang X, Cheng Z, Tang Y, Tao J. Multi-walled carbon nanotubes prevent high temperature-induced damage by activating the ascorbate-glutathione cycle in Paeonia ostii T. Hong et J. X. Zhang. Ecotoxicol Environ Saf. 2021;227:112948. 10.1016/j.ecoenv.2021.112948.

70. Kumari A, Gupta AK, Sharma S, Jadon VS, Sharma V, Chun SC, et al. Nanoparticles as a tool for alleviating plant stress: Mechanisms, implications, and challenges. Plants. 2024;13:1528. 10.3390/plants13111528.

71. Priyanka N, Venkatachalam P. Biofabricated zinc oxide nanoparticles coated with phycomolecules as novel micronutrient catalysts for stimulating plant growth of cotton. Adv Nat Sci NanoSci NanoTechnol. 2016;7:045018. 10.1088/2043-6262/7/4/045018. 72. Rajput VD, Minkina T, Kumari A, Harish, Singh VK, Verma KK, et al. Coping with the challenges of abiotic stress in plants: New dimensions in the field application of nanoparticles. Plants. 2021;10:1221. 10.3390/plants10061221.

73. Liu Y, Cao X, Yue L, Wang C, Tao M, Wang Z, et al. Foliar-applied cerium oxide nanomaterials improve maize yield under salinity stress: Reactive oxygen species homeostasis and rhizobacteria regulation. Environ Pollut. 2022;299:118900. 10.1016/j.envpol.2022.118900.

74. Ulhassan Z, Ali S, Kaleem Z, Shahbaz H, He D, Khan AR, et al. Effects of Nanosilica priming on rapeseed ( Brassica napus ) tolerance to cadmium and arsenic stress by regulating cellular metabolism and antioxidant defense. J Agric Food Chem. 2025;73:4518–33. 10.1021/acs.jafc.4c08246.

Associated Data Supplementary Materials Supplementary Material 1: Table S1. Impact of glutathione-gold nanoclusters (GSH-Au NCs) on the growth and biomass of vegetable sweet potato under normal and heat stress conditions. Table S2. List of the primers of genes encoding GSH-AsA cycle enzymes and antioxidant POD and GST enzymes. Table S3. Table of Pearson’s correlation analysis. Fig. S1. Characterization of GSH-Au nanoclusters (GSH-Au NCs). Fig. S2. Impact of glutathione-gold nanoclusters (GSH-Au NCs) on the root traits of vegetable sweet potato under normal and heat stress conditions. Data Availability Statement Data will be made available on request.

📖 中文全文 Chinese Full Text

中文

59 bmcps BMC Plant Biology BMC Plant Biol BMC PMC13059601 13059601 13059601 41772431 10.1186/s12870-026-08417-w 谷胱甘肽-金纳米簇通过改善光合作用、氧化还原稳态和细胞完整性增强甘薯耐热性 Kumar Sunjeet 1 2 # Gurmendar 1 2 # Yu Rui 1 2 Ikram Muhammad 1 2 Kou Jingjing 1 2 Khan Muhammad Abbas 1 2 Wang Mengzhao 1 2 ✉ Zhu Guopeng 1 2 ✉ 1 海南大学繁育与多倍体育种学院(三亚繁育与多倍体育种研究所),三亚,572025,中国 2 海南省热带园艺作物品质调控重点实验室,海南大学热带农林学院,海口,570228,中国 ✉ 通讯作者。# 共同第一作者。 2 3 2026 26 627 627 9 4 2026 © 作者 2026 开放获取 本文采用知识共享署名-非商业性使用-禁止演绎 4.0 国际许可协议授权,允许在任何媒介或格式下进行非商业性使用、共享、分发和复制,前提是给予原作者和来源适当的署名,提供知识共享许可协议的链接,并说明是否对许可材料进行了修改。未经本许可协议授权,您不得共享改编自本文或其部分的改编材料。本文中包含的图像或其他第三方材料均包含在文章的知识共享许可协议中,除非在材料的信用额度中另有说明。如果材料未包含在文章的知识共享许可协议中,且您的预期使用不被法定法规允许或超出允许的使用范围,您需要直接从版权所有者处获得许可。要查看本许可协议的副本,请访问 http://creativecommons.org/licenses/by-nc-nd/4.0/ 。

**摘要**

**背景** 全球变暖通过破坏生理、生化和结构过程,严重挑战甘薯种植。谷胱甘肽-金纳米簇(GSH-Au NCs)在增强耐热性方面的潜力在作为重要工业和粮食安全作物的菜用甘薯中尚未得到充分探索。我们假设叶面喷施GSH-Au NCs将通过增强抗氧化防御和酶活性,缓解热胁迫对光合作用、氧化还原稳态和细胞结构的破坏。

**结果** 在受控热胁迫条件下(42°C白天/35°C夜间,持续7天),热胁迫降低了茎生物量、叶面积、相对含水量(RWC)和根系生物量,同时损害光合作用并增加氧化损伤(H₂O₂:276.7%;MDA:481.5%)和电解质渗漏(85.7%)。GSH-Au NCs(2 mg L⁻¹)逆转了这些效应,促进了生长,提高了茎和根生物量(分别为45.3%和28.3%)以及RWC(7.9%)。这些改善与光合效率的提高相关,表现为叶绿素含量(72.6%)、Rubisco活性(26.4%)、气体交换参数(Pn:86.1%;Gs:389%)和叶绿素荧光(Fv/Fm:16.0%;ETR:48.2%)的升高。机制上,GSH-Au NCs与抗坏血酸-谷胱甘肽(AsA-GSH)循环的上调相关,增强了酶促(SOD:30.1%;APX:33.6%;GR:20.9%)和非酶促抗氧化剂(AsA/DHA:33.8%;GSH/GSSG:29.1%),同时降低了氧化标志物:H₂O₂(37.8%)、MDA(51.7%)和EL(26.8%),与未经纳米簇处理的胁迫植株相比。SOD、APX、GR、DHAR和MDHAR基因的转录上调支持了这些效应。此外,GSH-Au NCs增强了乙二醛酶活性(Gly I:36.9%;Gly II:35.0%),降低了有毒甲基乙二醛(29.3%)。较高的脯氨酸(51.7%)和次生代谢物(多酚:38.3%;类黄酮:62.9%)进一步增强了胁迫抗性。此外,GSH-Au NCs恢复了气孔行为,并保护了叶绿体和线粒体结构。

**结论** 这些发现强调了GSH-Au NCs(2 mg L⁻¹)作为一种可持续、经济、环保的纳米生物技术策略,用于缓解甘薯热胁迫并促进气候适应性园艺,通过首次证明其组合纳米簇形式的协同效应,超越了先前关于单独GSH或金纳米材料的研究。

**补充信息** 在线版本包含补充材料,获取地址为10.1186/s12870-026-08417-w。

**关键词:** 耐热性,GSH-Au NCs,氧化还原平衡,光合效率,AsA-GSH循环,超微结构状态

**引言**

气候变化驱动的全球变暖对植物健康和农业生产力构成严重威胁,因为温度升高和频繁的热浪对作物生产产生负面影响[1, 2]。热胁迫的特征是温度超过植物的最适生长范围,破坏细胞稳态,损害关键生理过程,降低作物产量,对植被产生长期后果[2, 3]。研究预测全球气温将持续上升,每升高1°C可能导致作物产量下降3-8%,特别是在全球70%人口居住的热带和亚热带地区[4, 5]。作为主要的农业生产国,中国面临日益严重的热浪威胁,热浪通过损害光合作用、水分调节和细胞完整性等关键过程,危及粮食安全[6-8]。

热胁迫深刻影响植物生理,特别是损害光合效率[9]。温度升高诱导气孔关闭,从而限制CO₂吸收并降低Rubisco羧化效率,同时促进光呼吸[10]。虽然气孔关闭有助于保水,但也加剧了CO₂限制,进一步降低光合效率。Rubisco等关键光合酶失活、叶绿素降解和叶绿体结构损伤加剧了这些效应。此外,光合传递链中过量的电子流,特别是光系统II(PSII)和PSI之间,增加了活性氧(ROS)的产生,包括超氧阴离子(O₂•⁻)、过氧化氢(H₂O₂)和甲基乙二醛(MG)。ROS水平升高导致氧化应激、脂质过氧化和膜损伤[6, 10, 11]。丙二醛(MDA)是脂质过氧化的标志物,在热胁迫下升高,表明叶绿体和线粒体等细胞结构受损。此外,根系呼吸和韧皮部运输受限阻碍了糖的转运,导致叶片中碳水化合物积累,进一步抑制CO₂同化[8, 10]。

植物通过防御系统对抗热胁迫,包括酶促清除剂,如超氧化物歧化酶(SOD)、过氧化氢酶(CAT)、过氧化物酶(POD)和抗坏血酸过氧化物酶(APX),以及非酶促化合物如谷胱甘肽(GSH)和脯氨酸。抗坏血酸-谷胱甘肽(AsA-GSH)循环对ROS解毒至关重要,利用AsA作为电子供体中和H₂O₂,产生单脱氢抗坏血酸(MDHAR)和脱氢抗坏血酸(DHAR)。GR利用NADPH从GSSG再生GSH,维持循环效率[12, 13]。此外,有毒化合物MG通过乙二醛酶(Gly I和II)的协同作用解毒[11]。这些防御机制使耐热品种能够维持细胞氧化还原平衡并保护其光合机构,而敏感品种通常经历严重的细胞损伤和加速的早衰[3]。长期胁迫耗竭抗氧化防御系统,使其失效。此时,施用外源生长调节剂可以改善胁迫抗性和恢复。

缓解热胁迫的传统方法,如化学处理和农艺措施,通常存在环境毒性、高成本和土壤微生物群落风险等局限性[14]。相比之下,纳米技术已成为一种有前景的环保解决方案,提供改善的营养递送、增强的胁迫抗性和可持续的作物保护[15, 16]。各种纳米材料(NMs),如纳米簇(NCs)和纳米颗粒(NPs),包括Au、SiO₂、CeO₂和MT-Se,通过增强生物量积累、营养吸收、光合效率和ROS清除来改善植物胁迫抗性[17-20]。这些纳米材料影响细胞结构、生理过程以及与水通道蛋白、光合作用和抗氧化防御相关的基因表达[21, 22]。

谷胱甘肽(GSH)是解毒、细胞分裂和胁迫抗性的关键调节因子,在热胁迫和其他非生物胁迫条件下增强抗氧化酶活性并保护光合过程[23, 24]。虽然金(Au)纳米材料和GSH补充剂在增强植物胁迫抗性方面各自显示出前景,但谷胱甘肽-金纳米簇(GSH-Au NCs)的组合应用尚未在任何植物物种中报道。选择GSH-Au NCs是因为其潜在的协同效应,其中GSH的抗氧化和解毒作用(如ROS清除和AsA-GSH循环调节)通过金纳米簇的生物物理特性得到增强,如高表面积用于有效递送、自由基清除和胁迫反应基因上调,如先前Au NP研究中观察到的[25-27]。这种组合旨在比单独组分提供更有针对性和稳定的相互作用。

本研究解决了先前纳米技术缓解热胁迫研究中的关键空白,这些研究集中在不同作物中的单独纳米材料,如Au NPs和GSH单独使用[17-20],但缺乏组合的GSH-Au NC配方、在菜用甘薯中的应用,以及对超微结构、转录和乙二醛酶反应的详细机制见解。选择甘薯(Ipomoea batatas)是因为其对热胁迫高度敏感,如在超过35°C温度下生物量和产量降低[13, 28, 29]。甘薯具有重要的农艺价值,是全球第七大经济重要的淀粉作物(全球年产量9350万公吨,中国单独占全球产量的55%)FAO, 2023, https://www.fao.org/faostat/en/#data/QCL/visualize)。先前关于热胁迫下甘薯的研究主要集中在基因表达、储藏根发育和遗传转化,对菜用甘薯的研究有限[28-31],并且缺乏该物种在热胁迫下基于纳米材料的胁迫缓解研究。

因此,我们假设叶面喷施GSH-Au NCs将通过以下方式增强甘薯的耐热性:(i)通过增加色素含量、Rubisco活性和气体交换来提高光合效率,(ii)通过激活AsA-GSH循环、乙二醛酶系统、抗氧化防御和相关基因表达来增强氧化还原平衡,以及(iii)保持结构完整性,包括气孔行为和叶绿体超微结构。研究结果旨在确立GSH-Au NCs作为一种经济、环保和可持续的方法,改善甘薯在热胁迫下的生长和抗性,为气候适应性农业提供新见解。

**材料与方法**

**购买纳米簇的表征**

分析纯GSH-Au NCs购自XFNANO Materials Technology Co., Ltd.(中国江苏),纯度高于99.5%。使用HR-TEM(S-4800, Hitachi, Tokyo, Japan)对GSH-Au NCs进行表征。纳米材料悬浮液在黑暗中超声处理30分钟,然后使用Xetasizer Nano ZS90(Malvern Instruments, Marburg, UK)计算zeta电位、紫外-可见吸收光谱和粒径分布[6]。GSH-Au NCs呈现球形形态,平均粒径<3 nm(HR-TEM),正zeta电位(22.7 ± 1.2 mV,确认胶体稳定性),以及在418 nm处的紫外-可见吸收峰(图1;S1)。

**图1** 谷胱甘肽-金纳米簇(GSH-Au NCs)的表征以及正常和热胁迫条件下的生长参数。A GSH-Au NCs的HR-TEM图像(比例尺:2 nm),B GSH-Au NCs的zeta电位,C 不同处理下菜用甘薯的代表性表型(CK:对照,GA:2 mg L⁻¹ GSH-Au NCs,HS:热胁迫,GAHS:2 mg L⁻¹ GSH-Au NCs + 热胁迫),D-K)定量生长参数:D 株高(cm plant⁻¹),E 叶面积(cm²),F 叶片数 plant⁻¹,G 相对含水量,H 茎鲜重(g plant⁻¹),I 茎干重(g plant⁻¹),J 根鲜重(g plant⁻¹),K 根干重(g plant⁻¹)。数据为三次生物学重复的平均值±SE。不同字母表示显著差异(Duncan检验,P < 0.05)。

**植物材料准备和实验设计**

实验使用甘薯品种'HD7791',该品种由我们研究组从中国海南省栽培群体中选育和评估。本研究的种植材料(茎插条)来自我们的机构库存。该品种的身份由我们研究团队正式确认,资源保存在海南大学。茎插条用杀菌剂消毒,在25-27°C、16小时光周期的土壤填充盆中生长。经过7天适应期后,生长室设置为42°C(光照)和35°C(黑暗),光照强度4,000 lx(80 µmol m⁻² s⁻¹ PPFD,基于LED转换因子),16/8小时光周期,湿度75%。选择这种中等光照水平是为了在7天急性热胁迫暴露期间维持一致的营养条件,允许清晰检测热诱导的生理破坏(光合作用下降和氧化损伤),而不会因高光光抑制而混淆。这种制度模拟了热带和亚热带甘薯种植区的极端热浪,夏季温度经常超过42°C,导致20-40%的产量损失[4, 5, 28]。

叶面喷施在定义的早期营养阶段(适应后7天)开始,此时在预试验中所有植株均匀出现初始热胁迫表型(轻度萎蔫),确保时间一致并减少偏差。GSH-Au NCs的选择浓度(0、0.5、1、2和4 mg L⁻¹)基于初步优化和先前报道的纳米材料介导胁迫缓解的生理有效浓度,无植物毒性效应。

实验设计包括以下处理:(1)CK;对照,(2)GA1;0.5 mg L⁻¹ GSH-Au NCs,(3)GA2;1 mg L⁻¹ GSH-Au NCs,(4)GA3;2 mg L⁻¹ GSH-Au NCs,(5)GA4;4 mg L⁻¹ GSH-Au NCs,(6)HS;热胁迫42°C,(7)GAHS1;0.5 mg L⁻¹ GSH-Au NCs + 热胁迫,(8)GAHS2;1 mg L⁻¹ GSH-Au NCs + 热胁迫,(9)GAHS3;2 mg L⁻¹ GSH-Au NCs + 热胁迫,(10)GAHS4;4 mg L⁻¹ GSH-Au NCs + 热胁迫。在NC处理7天后记录表型和生理数据。

**植物生长参数的测定**

评估甘薯幼苗的生长参数,包括高度、叶片数、叶面积以及根和茎的鲜重和干重。叶面积使用便携式激光叶面积仪(CI-202, CID Bio-Science, USA)测定。记录鲜重(FW),然后在80°C干燥72小时以测定干重(DW)。相对含水量(RWC)使用既定方法计算[24]。在测定叶片FW后,将其浸入ddH₂O中4小时,然后测定其膨胀重量(TW)。然后将叶片在70°C干燥24小时。最后,使用以下公式测量叶片的RWC;RWC (%) = [(FW-DW)/(TW-DW)] × 100。

清洗后,使用Epson Expression 11000XL根扫描仪扫描新鲜根以确定根形态。使用WinRHIZO 2003a软件分析图像,这是近期植物研究中根性状的验证和可靠工具[32]。

**光合作用相关参数的测定**

使用便携式光合系统测量叶片气体交换参数。叶绿素和类胡萝卜素含量通过将0.1 g新鲜叶片在80%丙酮中匀浆,然后离心并在662、645和470 nm处测量吸光度来测定。使用TCS SP2激光共聚焦显微镜(Leica, Germany)进行叶绿素和类胡萝卜素可视化。使用便携式叶绿素荧光仪定量ETR、qP、qN、Y(II)和Fv/Fm[32, 33]。

**电子显微镜分析**

对于扫描电子显微镜(SEM),用蒸馏水冲洗新鲜叶片,用戊二醛固定,并在80%乙醇中脱水。然后使用临界点干燥干燥样品,用导电胶带固定在SEM桩上,并通过溅射镀铂10分钟,然后进行SEM成像[34]。

对于透射电子显微镜(TEM),将新鲜叶组织切成小块,在4°C下用2.5%戊二醛固定12小时。样品在0.1 M磷酸盐缓冲盐水(PBS, pH 7.4)中洗涤三次,每次15分钟,在1%锇酸中后固定1-2小时,再在PBS中洗涤三次,每次15分钟。使用分级乙醇系列(30%、50%、70%和90%各15分钟,然后100%乙醇两次各20分钟)和两次100%丙酮各20分钟进行脱水。用丙酮:包埋树脂混合物(1:1在37°C 3小时和1:3在37°C 4小时)进行浸润,然后在37°C下用纯包埋树脂过夜。随后将样品包埋在新鲜树脂模具中,在70°C下聚合12-48小时。切割超薄切片,安装在铜网上,用醋酸铀染色8-15分钟,然后用柠檬酸铅染色5-10分钟,风干,并在HITACHI HT7800 TEM下检查[24]。

**通过组织化学定位检测氧化损伤**

H₂O₂和O₂•⁻分别使用DAB和NBT染色进行定位。MDA通过将新鲜叶片用10%希夫试剂染色120分钟,然后在偏亚硫酸氢钾溶液中洗涤以去除残留染料来检测[35]。

**氧化应激标志物、抗氧化剂和抗氧化酶的测定**

电解质渗漏(EL)使用先前建立的方案测量[36]。氧化应激标志物(H₂O₂、O₂•⁻和MDA)、抗氧化剂(AsA、DHA、GSH和GSSG)、Rubisco活性和抗氧化酶活性(SOD、CAT、APX、GR、DHAR、MDHAR、POD和GST)的水平使用南京建成生物工程公司和Solarbio的商业试剂盒定量。将叶片样品匀浆,离心,按照试剂盒说明分析上清液[13, 24]。MG含量和Gly I和Gly II的活性使用先前描述的方案测量[36]。

**渗透调节物质和次生代谢物含量的测定**

可溶性糖通过将叶片样品在双蒸水中匀浆,在95°C加热15分钟,并在8,000 g离心12分钟来提取。使用中国南京建成生物工程公司的商业测试试剂盒在620 nm处测量吸光度。总蛋白和脯氨酸浓度使用商业试剂盒(A0452和A107-1-1)在匀浆和离心后定量[13, 24]。使用Folin-Ciocalteu法测定总多酚,使用氯化铝法测定类黄酮含量[24, 37]。

**qRT-PCR分析**

使用Trizol试剂盒(Invitrogen, Santa Cruz, CA, USA)从冷冻叶片样品中提取总RNA。使用SuperScript III逆转录酶试剂盒进行逆转录。Actin基因(登录号:EU250003)用作归一化的内参,基于其在处理间的稳定表达选择。使用Primer3软件设计基因特异性引物(引物序列列于表S2)。使用Bio-Rad Mx3000P qPCR系统进行qRT-PCR分析,三次生物学重复(n = 3个独立植株/处理)。使用2⁻ΔΔCt方法计算表达水平[38, 39]。

**统计分析**

所有实验测量均进行三次生物学重复(n = 3个独立植株/处理)。数据表示为平均值±标准误差(SE)。使用SPSS 25.0(IBM, Chicago, USA)中的单因素方差分析(ANOVA)和Duncan多重范围检验在P < 0.05下分析处理间的统计差异。选择Duncan检验是因为其在多处理农业实验中检测差异的敏感性,这在植物胁迫研究中常用。为了最小化与Duncan检验相关的I型错误膨胀风险,所有关键结果均与HSD检验(P < 0.05)交叉验证,显示显著性模式无差异。事后确认了主要参数的统计功效(功效>0.8)。使用R软件(ggcor包)进行Pearson相关分析。

**结果**

**植物生长变量**

在正常和热胁迫条件下测试了不同浓度,以研究GSH-Au NCs对甘薯植株的生物学影响。在非胁迫条件下,与对照植株相比,GSH-Au NCs(GA1至GA4)导致生长和生物量积累显著增强(P < 0.05),其中GA3(2 mg L⁻¹)处理显著促进生长,茎和根鲜生物量分别增加13.5%和12.1%,干生物量分别增加7.2%和9.2%,光合色素增加3.6-7.2%(表S1;图2),表明除胁迫缓解外的一般促生长效应。

**图2** 正常和热胁迫条件下甘薯叶片的自发荧光、叶绿素和类胡萝卜素含量。A 叶绿素和类胡萝卜素的自发荧光,B-E 正常和热胁迫条件下的叶绿素和类胡萝卜素含量(mg g⁻¹ FW)。处理:CK = 对照,GA = 2 mg L⁻¹ GSH-Au NCs,HS = 热胁迫,GAHS = 2 mg L⁻¹ GSH-Au NCs + 热胁迫。数据为n = 3次生物学重复的平均值±SE。不同字母表示显著差异(Duncan检验,P < 0.05)。

然而,热胁迫植株的发育严重受损,与对照植株相比,株高(24.1%)、茎FW和DW(41.6和43.1%)、叶片数(56.5%)、叶面积(66.7%)、RWC(15.1%)、根生物量(40.4% FW和31.5% DW)降低(图1,表S1)。值得注意的是,GSH-Au NCs缓解了这些不利影响,2 mg L⁻¹(GAHS3)处理显示出最高效力,与热胁迫植株相比,株高(18.9%)、茎生物量(45.3% FW,35.2% DW)、叶片性状(叶片数增加100%,叶面积增加95.7%)、RWC(7.9%)和根生物量(28.3% FW和25.6% DW)增强(图1,表S1)。基于这些结果,2 mg L⁻¹ GSH-Au NCs浓度被确定为最佳处理,并选择用于进一步分析。

根系构型也受到热胁迫的严重影响(P < 0.05;图S1)。然而,用2 mg L⁻¹ GSH-Au NCs处理的甘薯植株在根性状方面表现出显著恢复,包括根长(27.9%)、表面积(68.1%)、直径(12.7%)、根体积(59.9%)、投影面积、尖端、分叉和交叉点与热胁迫植株相比显著增加(图S2)。

**光合色素、Rubisco活性和光合效率**

在正常条件下,GSH-Au NCs轻微增强了光合色素水平和效率(图2和3)。然而,热胁迫暴露使T. Chl、Chl a、Chl b和Car水平分别大幅降低63.7%、53.5%、74.5%和46.6%(图2)。而GSH-Au NCs(2 mg L⁻¹)在热胁迫下显著增加色素浓度,与热胁迫植株相比,T. Chl增加72.6%,Chl a增加69.2%,Chl b增加79.1%,Car增加28.2%(图2 B-E)。共聚焦显微镜显示,在正常条件下,对照和NC处理植株中红色(叶绿素)和绿色(类胡萝卜素)荧光均匀且密集。然而,热胁迫叶片中叶绿素和类胡萝卜素荧光急剧下降。GSH-Au NCs的施用恢复了荧光强度,与观察到的色素恢复一致(图2 A)。

**图3** 正常和热胁迫条件下菜用甘薯的叶绿素荧光、气体交换参数和Rubisco活性。A-E Fv/Fm、Y(II)、qP和ETR。F-I Pn、Gs、Ci和Tr。J 正常和热胁迫条件下的Rubisco活性。处理:CK = 对照,GA = 2 mg L⁻¹ GSH-Au NCs,HS = 热胁迫,GAHS = 2 mg L⁻¹ GSH-Au NCs + 热胁迫。数据为n = 3次生物学重复的平均值±SE。不同字母表示显著差异(Duncan检验,P < 0.05)。

关键的碳固定酶Rubisco在热胁迫下活性降低38.3%,成为光合效率的主要限制因素。然而,GSH-Au NCs显著缓解了热诱导的Rubisco抑制,与胁迫植株相比,活性恢复26.4%(图3 J)。这种增强与气体交换显示的改善的碳同化速率直接相关。

反映光系统II(PSII)功能状态的叶绿素荧光参数也受到热胁迫的严重影响。Fv/Fm、Y(II)、qP和ETR分别降低25.8%、58.2%、56.3%和55.7%,表明光能转换受损(图3 A-E)。GSH-Au NCs处理抵消了这些负面影响,Fv/Fm提高16.0%,Y(II)提高93.6%,qP提高71.3%,ETR提高48.2%(图3 A-E),从而改善胁迫下的所有光合效率。

气体交换测量进一步支持了这些发现。与对照植株相比,热胁迫导致Pn(57.1%)、Gs(87.7%)、Ci(47.0%)和Tr(66.1%)大幅降低。然而,GSH-Au NC处理的植株表现出显著恢复,Pn增加86.1%,Gs增加389.0%,Ci增加47.3%,Tr增加124.3%(图3 F-I)。这些结果表明,GSH-Au NCs不仅在热胁迫下保护光合机构,还增强气孔功能和碳同化。

**气孔参数**

热胁迫严重影响甘薯叶片的气孔特征,降低密度(图4 A)和尺寸,包括长度(61.1%)、宽度(75.3%)和孔长度(61.0%)。GSH-Au NCs处理缓解了这些形态变化,与热胁迫植株相比,气孔长度增加90.7%,宽度增加161.4%,孔长度增加60.9%(图4 B-E)。GSH-Au NCs促进气孔开放并增加气孔密度,表明在胁迫条件下气体交换能力改善。

**图4** 正常和热胁迫条件下菜用甘薯的气孔特征。A 250倍放大下的气孔密度,比例尺200 μm,B 3000倍放大下的气孔,比例尺10 μm,C 气孔长度,D 气孔宽度,E 正常和热胁迫条件下的气孔孔长度。处理:CK = 对照,GA = 2 mg L⁻¹ GSH-Au NCs,HS = 热胁迫,GAHS = 2 mg L⁻¹ GSH-Au NCs + 热胁迫。数据为n = 3次生物学重复的平均值±SE。不同字母表示显著差异(Duncan检验,P < 0.05)。

**叶绿体超微结构分析**

TEM揭示了正常和热胁迫条件下甘薯叶肉细胞的结构差异。在正常条件下,对照和GSH-Au NCs处理的叶肉细胞均保持均匀形状,叶绿体呈现椭圆形结构和完整的类囊体膜。基质清晰可见,细胞壁薄且边缘清晰,基粒片层紧密堆积,基粒和基质片层组织良好。线粒体保持其结构完整性,表明呼吸正常(图5)。

**图5** 正常和热胁迫条件下菜用甘薯的超微结构变化。CK = 对照,GA = GSH-Au NCs,HS = 热胁迫,GAHS = 热胁迫 + GSH-Au NCs。缩写:CW = 细胞壁,Chl = 叶绿体,M = 线粒体,C = 细胞质,S = 淀粉,SL = 基质片层,G = 基粒,PG = 质体小球。叶肉细胞4000倍放大,比例尺2 μm。叶绿体结构15,000倍放大,比例尺1 μm。

热胁迫诱导显著的超微结构变化;叶肉细胞肿胀,叶绿体呈现异常肿胀、类囊体破坏和基质不规则,自噬囊泡增加表明强烈的自我消化。细胞壁发育受损,边缘模糊,而基粒片层分离,线粒体出现损伤(图5)。

相比之下,热胁迫下GSH-Au NCs处理的植株显示细胞结构部分恢复。细胞大小接近对照水平,叶绿体肿胀减少,类囊体结构部分恢复,自噬活动减少。细胞壁边缘恢复清晰,基粒和基质片层显示改善的组织,线粒体肿胀不那么明显(图5)。这些变化突出了GSH-Au NCs在热胁迫条件下维持叶绿体超微结构的保护作用。

**氧化损伤、渗透调节物质和非酶促抗氧化剂**

在热胁迫下,甘薯叶片表现出显著的氧化损伤,表现为ROS如H₂O₂和O₂•⁻水平升高,以及脂质过氧化(MDA)和EL增加。组织化学染色揭示了明显的视觉标志物,如H₂O₂的棕色斑点、O₂•⁻的蓝色斑点和MDA的品红色,显示其在热胁迫下的积累。然而,GSH-Au NCs处理通过减少斑点的强度和数量有效缓解了这些效应(图6 A)。

浓度测定还显示,与对照植株相比,热胁迫植株的H₂O₂含量增加276.7%,O₂•⁻增加143.1%,MDA增加481.5%,EL增加85.7%(P < 0.05;图6 B-E)。值得注意的是,热胁迫下GSH-Au NCs的施用显著缓解了氧化损伤,与热胁迫植株相比,H₂O₂减少37.8%,O₂•⁻减少36.6%,MDA减少51.7%,EL减少26.8%(图6 B-E)。

**图6** 正常和热胁迫条件下菜用甘薯的氧化损伤、渗透调节物质和非酶促抗氧化剂。A 组织化学定位。B-E H₂O₂、O₂•⁻、MDA和电解质渗漏(EL),F 脯氨酸,G 可溶性糖,H 总多酚,I 总类黄酮在正常和热胁迫条件下。处理:CK = 对照,GA = 2 mg L⁻¹ GSH-Au NCs,HS = 热胁迫,GAHS = 2 mg L⁻¹ GSH-Au NCs + 热胁迫。数据为n = 3次生物学重复的平均值±SE。不同字母表示显著差异(Duncan检验,P < 0.05)。

为了克服氧化胁迫,甘薯植株积累渗透调节物质(脯氨酸和可溶性糖)和非酶促抗氧化剂(多酚和类黄酮)。热胁迫触发显著增加,与对照植株相比,脯氨酸增加157.4%,可溶性糖增加192.1%,多酚增加167.8%,类黄酮增加239.4%(图6 F-I)。GSH-Au NCs进一步增强这种适应性反应,与热胁迫植株相比,脯氨酸(51.7%)、多酚(38.3%)和类黄酮(62.9%)显著增加。有趣的是,GSH-Au NCs在热胁迫下降低可溶性糖20.1%(图6 F-I)。

**抗坏血酸、谷胱甘肽及其比率**

本研究观察到热胁迫下甘薯植株AsA-GSH循环的显著变化。具体而言,热胁迫导致AsA水平显著增加57.3%,而DHA含量降低11.9%,导致与对照条件相比AsA/DHA比率显著增强78.1%。当在热胁迫下施用GSH-Au NCs时,AsA含量进一步增加17.9%,DHA水平额外降低11.2%。这导致AsA/DHA比率增加33.8%(图7 A-C)。

**图7** 抗坏血酸、谷胱甘肽及其比率含量。A AsA,B DHA,C AsA/DHA比率,D GSH,E GSSG,F 正常和热胁迫条件下的GSH/GSSG比率。处理:CK = 对照,GA = 2 mg L⁻¹ GSH-Au NCs,HS = 热胁迫,GAHS = 2 mg L⁻¹ GSH-Au NCs + 热胁迫。数据为n = 3次生物学重复的平均值±SE。不同字母表示显著差异(Duncan检验,P < 0.05)。

同样,GSH、GSSG水平及其相应的GSH/GSSG比率在热胁迫下也显著升高,与对照植株相比分别增加150.6%、76.0%和43.0%。热胁迫下GSH-Au NCs的施用进一步放大了这些效应,GSH增加56.0%,GSSG增加20.9%,GSH/GSSG比率增加29.1%(图7 D-F)。

**抗氧化酶(AsA-GSH循环)**

热胁迫暴露显著增强了甘薯中几种关键酶的活性。与对照植株相比,SOD、CAT、APX、GR、DHAR和MDHAR活性分别增加89.2%、63.6%、414.4%、216.7%、237.1%和126.8%。热胁迫下GSH-Au NCs的施用进一步提高了除CAT外所有上述酶的活性。具体而言,GSH-Au NCs处理使SOD增加30.1%,APX增加33.6%,GR增加20.9%,DHAR增加54.9%,MDHAR增加46.6%。令人惊讶的是,GSH-Au NCs处理后CAT活性降低20.5%(图8 A-F)。

**图8** 正常和热胁迫条件下的抗氧化酶活性。A-F GSH-AsA循环酶(SOD、CAT、APX、GR、DHAR和MDHAR)。G-H POD和GST活性。处理:CK = 对照,GA = 2 mg L⁻¹ GSH-Au NCs,HS = 热胁迫,GAHS = 2 mg L⁻¹ GSH-Au NCs + 热胁迫。数据为n = 3次生物学重复的平均值±SE。不同字母表示显著差异(Duncan检验,P < 0.05)。

POD和GST在清除H₂O₂和解毒脂质过氧化物方面发挥关键作用。热胁迫使POD和GST活性分别升高197.0%和83.8%。然而,GSH-Au NCs处理在热胁迫条件下进一步使POD增加33.5%,GST增加53.6%(图8 G-H)。

**基因的转录表达**

在热胁迫条件下,甘薯植株中多个抗氧化酶相关基因的转录水平显著上调,除了CAT显示下调。值得注意的是,SOD、APX、GR、DHAR和MDHAR显示出转录活性显著升高(图9 A-F)。GSH-Au NCs处理进一步增强了这些上调基因在热胁迫植株中的表达,而CAT表达保持抑制(图9 A-F),与观察到的酶活性模式一致。

同样,POD和GST表达水平在热胁迫下增加。然而,GSH-Au NCs处理导致POD水平非显著降低,而GST表达在热胁迫条件下进一步增强(图9 G)。

**图9** 热胁迫条件下编码抗氧化酶的基因的转录表达。A-F 编码GSH-AsA循环酶的基因(SOD、CAT、APX、GR、MDHAR和DHAR)。G,H POD和GST基因。处理:CK = 对照,HS = 热胁迫,GAHS = 2 mg L⁻¹ GSH-Au NCs + 热胁迫。数据为n = 3次生物学重复的平均值±SE。不同字母表示显著差异(Duncan检验,P < 0.05)。

**甲基乙二醛和乙二醛酶活性**

热胁迫显著增加了甘薯植株中MG的积累,与对照植株相比,MG水平增加137.5%。然而,热胁迫下GSH-Au NCs的施用有效降低MG含量29.3%。相反,热胁迫抑制Gly I和Gly II的活性,与对照植株相比分别降低37.7%和40.3%。热胁迫条件下GSH-Au NCs的施用显著恢复其活性,Gly I和Gly II分别增加36.9%和35.0%(图10 A-C)。

**图10** 正常和热胁迫条件下的甲基乙二醛和乙二醛酶。A 甲基乙二醛(MG)含量,B 乙二醛酶I(Gly I),C Gly II活性。处理:CK = 对照,GA = 2 mg L⁻¹ GSH-Au NCs,HS = 热胁迫,GAHS = 2 mg L⁻¹ GSH-Au NCs + 热胁迫。数据为n = 3次生物学重复的平均值±SE。不同字母表示显著差异(Duncan检验,P < 0.05)。

**Pearson相关分析**

Pearson相关矩阵显示热胁迫甘薯植株关键生理参数之间的显著关系。结果表明,在热胁迫条件下,氧化应激标志物(MDA、H₂O₂、O₂•⁻和MG)、EL和可溶性糖积累之间存在强正相关。相比之下,GSH-Au NCs处理显示与这些胁迫指标呈显著负相关(图11;表S3),表明其对氧化损伤的保护作用。

此外,GSH-Au NCs与生长和生理参数呈正相关,如植株生物量、气体交换参数、叶绿素和类胡萝卜素含量、叶绿素荧光、Rubisco活性和抗氧化防御系统组分(除CAT外)。此外,GSH-Au NCs与乙二醛酶、次生代谢物积累、脯氨酸含量和气孔形态性状之间观察到正相关(图11;表S3)。总之,这些结果突出了GSH-Au NCs在增强抗氧化防御机制和提高甘薯植株耐热性方面的功效。

**图11** 热胁迫下GSH-Au NCs处理甘薯植株生长、生理生化参数的Pearson相关分析。给定参数的缩写与上述相同;SFW(茎鲜重),SDW(茎干重),RFW(根鲜重),RWC(相对含水量)等。在相关矩阵中,橙色线表示正相关,浅蓝色虚线表示负相关。

**讨论**

热胁迫损害甘薯生长、生物量、RWC和根系形态(图1;S2),可能是由于细胞分裂、代谢和光合作用受阻。这些变化阻碍营养和水分吸收,进一步加剧胁迫,如在各种植物胁迫条件下观察到的[10, 40, 41]。与传统集约农业不同,纳米技术使用纳米材料实现可持续生产,这些材料可能同时支持胁迫适应和营养吸收效率[15, 42]。

在本研究中,叶面喷施GSH-Au NCs(2 mg L⁻¹)与热胁迫效应的显著缓解相关,有助于甘薯植株茎和根生物量、株高、叶片发育和根系构型的恢复(图1;S2)。这些观察与纳米材料(Se、Ag和TiO₂)在热胁迫下改善番茄、小麦和菊花等作物的生长、生物量和RWC的研究一致[43-46]。

观察到的根性状恢复(图S2)可能有助于补偿胁迫诱导的光合产物不平衡,可能支持营养和水分吸收,通过增强膨压和细胞扩张促进植物生长[47, 48]。GSH-Au NCs还与RWC升高相关,可能与渗透调节物质积累,特别是脯氨酸有关。这种模式与碳纳米颗粒、AgNPs和ZnONPs的报告一致,这些物质在热胁迫条件下有助于改善细胞稳态、膜完整性和保水[45, 49, 50]。

这些反应表明,GSH-Au NCs可能支持生长、水分平衡、光合性能、代谢和结构完整性方面的胁迫缓解。光合作用对温度升高高度敏感,这可能损害光合机构的生化和结构组分[51]。在本研究中,热胁迫大幅降低气体交换参数(图3 F-I),可能限制CO₂吸收和蒸发冷却,如先前在番茄、高粱和菊花中报道的[52, 53]。

热胁迫还抑制叶绿素荧光(图3 A-E),表明可能的光系统II(PSII)损伤和电子传递受损,导致Fv/Fm比率降低并阻碍天线分子。这些变化可能是由于酶活性改变、保卫细胞功能障碍和ETC抑制[47]。因此,观察到甘薯叶片中Rubisco活性、叶绿素和类胡萝卜素含量以及强度降低(图2和3)。

然而,GSH-Au NCs与Gs、Tr、Pn和Ci改善相关,表明叶肉导度和碳同化增强。GSH-Au NCs处理还有助于叶绿素荧光参数(Fv/Fm、Y(II)、qP和ETR)的恢复,增加叶绿素和类胡萝卜素含量以及色素荧光强度(图2和3),并降低qN,表明PSII效率和光保护能力改善。这些反应与Au、TiO₂、MnO和Se纳米材料的报告一致,这些纳米材料与缓解光抑制、ROS清除以及维持番茄、香菜和小麦的气体交换相关[47, 54-57]。

Rubisco活性增强(图2 J)进一步支持Calvin-Benson循环调节的改善,与先前关于TiO₂和CeO₂的发现一致[58, 59]。这些改善与氧化应激减少一致,有助于在热胁迫下维持甘薯的光合性能和能量产生。

热胁迫可能损害气孔和超微结构,破坏气体交换和光合效率[10]。在本研究中,甘薯叶片显示气孔密度、长度、宽度和孔长度降低(图4),以及叶肉细胞、叶绿体和线粒体受损(图5)。这些结构改变可能阻碍光依赖反应和线粒体ATP合成[8, 52]。

GSH-Au NCs与改善的气孔性状(密度、长度、宽度和孔长度)相关(图4),可能促进气体交换、蒸发冷却和CO₂吸收。这些观察与CeO₂、ZnO和Zn-Si NPs增强高粱和豌豆气孔开放的报告一致[52, 60]。

电子显微镜显示,GSH-Au NCs与叶肉完整性、叶绿体结构(类囊体、基质和基粒)和线粒体形态的保持相关(图5),可能减少氧化损伤。保持的叶绿体可能支持光合作用,而完整的线粒体可能确保能量代谢。GSH-Au NC处理还似乎增加质体小球积累,这可能有助于脂质代谢和胁迫抗性。这些结构反应与小麦和水稻中AuNPs和SiO₂ NPs以及钴胁迫下CeO₂ NPs的先前研究一致[57, 61, 62]。

这些发现表明,GSH-Au NCs可能有助于维持气孔功能和超微结构,从而支持光合恢复。热胁迫触发过量ROS产生,导致氧化和羰基应激,可能损伤脂质、蛋白质和DNA[8, 11]。在本研究中,热胁迫甘薯植株显示H₂O₂、O₂•⁻、MDA、MG和EL升高,通过组织化学染色确认(H₂O₂、O₂•⁻和MDA;图6 A-E)。这些效应可能与光合作用下降和过量电子泄漏有关。

可溶性糖积累(图6 G)可能反映渗透调节,但也可能表明碳水化合物代谢紊乱[63]。GSH-Au NCs处理与氧化标志物(H₂O₂、O₂•⁻、MDA、MG和EL)减少和组织化学染色强度降低相关(图6 A-E)。这些发现与Se、S、CeO₂、ZnO和TiO₂纳米材料在非生物胁迫下抑制氧化损伤的先前研究一致[44, 47, 64]。

GSH-Au NCs还似乎降低可溶性糖水平,表明光合活性和碳水化合物代谢向生长和能量产生恢复[63, 65]。此外,GSH-Au NCs与乙二醛酶I和II活性增加相关(图10),可能通过GSH依赖性乙二醛酶途径促进MG解毒。这种模式与其他植物系统中纳米颗粒增强乙二醛酶活性的先前报告一致[47, 66]。

总之,这些协调反应表明,GSH-Au NCs可能通过多层抗氧化机制增强氧化还原稳态和耐热性。AsA-GSH循环在抗氧化防御中发挥核心作用,通过在热胁迫下解毒ROS和维持氧化还原平衡[67]。我们的结果表明,GSH-Au NCs与AsA和GSH水平增加、AsA/DHA和GSH/GSSG比率升高相关(图7 A-F),表明抗氧化能力增强。处理还上调AsA-GSH循环的酶活性(SOD、APX、GR、DHAR和MDHAR)和基因表达(图8和9)。

这些反应与碳纳米管、Se、S和MnO纳米材料增强牡丹、小麦和番茄中AsA-GSH组分的报告一致[47, 66, 68, 69]。这些AsA-GSH循环基因的上调可能涉及HSF、MYB和WRKY转录因子,可能由ROS或NP诱导的MAPK信号激活[50, 70]。

有趣的是,GSH-Au NCs与CAT活性降低相关(图8 B和9 B),可能是由于AsA-GSH途径活性增强导致H₂O₂负荷减少,与ZnO-NP研究一致[71, 72]。相比之下,POD和GST活性增加(图8 G-H)可能有助于过氧化物和外源物解毒[63, 66]。

GSH-Au NCs还与脯氨酸水平增强相关(图6 F),可能支持热胁迫下甘薯植株的渗透平衡、膜稳定性和ROS清除。这与各种纳米颗粒在非生物胁迫下增加脯氨酸积累的报告一致[54, 70]。

此外,GSH-Au NC处理有助于热胁迫下甘薯叶片中次生代谢物(多酚和类黄酮)增加(图6 H-I),可能通过苯丙烷途径促进ROS清除和光合恢复[13, 73]。这些观察与FeNPs、SeNPs、Ag NPs和SiNPs在各种植物中的效应一致[49, 63, 66, 74]。

总体而言,本研究表明GSH-Au NCs(2 mg L⁻¹)与甘薯植株热胁迫的显著缓解相关。机制上,GSH-Au NCs可能通过直接ROS清除(通过减少H₂O₂、O₂•⁻、MDA和EL证明)和AsA-GSH基因(SOD、APX、GR、MDHAR和DHAR)的转录上调来调节氧化还原平衡,与Au NP效应一致[25-27]。推测上,Au的催化性质可能增强GSH稳定性和再生,尽管酶动力学需要进一步确认。

与小麦中Au NPs(增强ROS清除)[26]类似,我们的结果显示氧化损伤减少;然而,GSH-Au NC协同作用独特地将GSH/GSSG提高29.1%,这是单独应用中未报道的反应,突出了新颖性,同时与番茄中GSH的作用一致[23]。此外,GSH-Au NC处理增加的乙二醛酶活性(Gly I和Gly II)有助于降低甲基乙二醛水平,进一步支持氧化还原稳态。

这些协调反应共同有助于生长、生物量、光合效率和细胞超微结构的恢复,同时通过ROS清除和AsA-GSH循环激活最小化氧化损伤。这些发现将GSH-Au NCs定位为非生物胁迫下作物抗性的有前景、可持续的解决方案。然而,局限性包括缺乏通过ICP-MS或TEM直接确认纳米簇吸收和分布、依赖可能不能完全反映田间变异或长期结果的短期温室评估,以及缺乏直接毒性分析。未来研究应优先考虑定位、扩展田间试验和多季节评估。

实际上,GSH-Au NCs为高温地区提供了一种低成本和环保的解决方案,但这些局限性需要谨慎解释。

**结论**

本研究首次提供了全面证据,表明GSH-Au NCs(2 mg L⁻¹)与菜用甘薯耐热性的显著增强相关,有助于改善生长、生物量、光合效率和结构完整性。这些效应与通过AsA-GSH循环激活、抗氧化酶活性和基因表达上调(SOD、APX、GR、DHAR、MDHAR、POD和GST)、乙二醛酶系统(Gly I和II)增强以及渗透调节物质(脯氨酸)积累而减少的氧化和羰基应激相关。GSH-Au NC处理还有助于气孔功能、叶绿体和线粒体超微结构的恢复,以及次生代谢物(多酚和类黄酮)增加,进一步支持抗性。

这些发现突出了GSH-Au NCs作为一种可持续、经济和环保的策略,用于缓解甘薯和潜在其他作物的热胁迫,通过证明其组合纳米簇协同效应,超越了先前关于单独GSH补充或金纳米材料的工作。然而,局限性包括缺乏直接的然而,需要进一步研究以确认直接吸收,阐明机制,在田间条件下验证这些发现,并评估纳米材料在农业作物系统中应用的长期环境影响和安全性。

**补充信息**

**补充材料1:** 表S1. 谷胱甘肽-金纳米簇(GSH-Au NCs)对正常和热胁迫条件下菜用甘薯生长和生物量的影响。表S2. 编码GSH-AsA循环酶和抗氧化POD和GST酶的基因引物列表。表S3. Pearson相关分析表。图S1. GSH-Au纳米簇(GSH-Au NCs)的表征。图S2. 谷胱甘肽-金纳米簇(GSH-Au NCs)对正常和热胁迫条件下菜用甘薯根性状的影响。

**致谢**

本研究由三亚崖州湾科技城"崖州湾"精英人才项目(SKJC-JYRC-2024-14;SKJC-JYRC-2024-25)、海南大学科研启动基金项目(XJ2400007871)、CARS-10-甘薯专项资金和海南省院士创新平台(YSPTZX202206)资助。我们也非常感谢国家热带植物种质资源中心。

**作者贡献**

Sunjeet Kumar:概念化、项目管理、资源、撰写-原稿、验证、方法、可视化。Gurmendar:调查、形式分析、撰写原稿。Rui Yu:调查。Muhammad Ikram:软件。Jingjing Kou:撰写-审阅和编辑。Muhammad Abbas Khan:撰写-审阅和编辑。Mengzhao Wang:可视化、项目管理、数据管理、验证。Guopeng Zhu:概念化、资源、监督、资金获取。

**数据可用性**

数据可根据要求提供。

**声明**

伦理批准和参与同意:不适用。发表同意:不适用。竞争利益:作者声明无竞争利益。

**脚注**

出版商说明:Springer Nature对已出版地图和机构隶属关系中的管辖权主张保持中立。Sunjeet Kumar和Gurmendar对本工作贡献相同。

**贡献者信息**

Mengzhao Wang,邮箱:mzwang@hainanu.edu.cn。Guopeng Zhu,邮箱:zhuguopeng@hainanu.edu.cn。